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Cavalier King Charles Spaniel

Cavalier King Charles Spaniel
Photo: Andreweatock / CC BY-SA 3.0 · Wikimedia

102 Cavalier King Charles Spaniels in the atlas. Every number on this page has a source.

Population-genetic snapshot of Cavalier King Charles Spaniels in the Sniff Atlas, source-graded Mendelian carrier frequencies from Donner 2023, and nutrition guidance tied to the genetic findings above.

Also known as CKCS.

The plain version

Cavalier King Charles Spaniels come from a gene pool that’s a bit limited, meaning they’re somewhat inbred. They’re a small, friendly breed, usually weighing around 15 lb and living about 10 years on average. Because of their genetic background, some dogs in this breed may carry genes linked to certain spine and nerve conditions, but this doesn’t mean every dog will have these issues. It’s a good idea to talk with your vet or consider genetic testing to keep your pup healthy.

What the atlas says about Cavalier King Charles Spaniel

In the atlas, the Cavalier King Charles Spaniel clusters consistently as Cavalier King Charles Spaniel (100% of the 102 dogs here). At the trait loci, FGF4_retrogene_CFA12 runs lower than average (0% here vs 80%); ADAMTS17 runs higher than the atlas average (97% here vs 54%).

Ranks 11 of 107 on the bottleneck severity scale, well into the upper quartile of population contraction.

Median lifespan is 10.0 years, about 3.0 years shorter than a typical dog of 6.7 kg, one of the larger gaps in the atlas.

Genetic dimensions · CanVAS atlas

What the genome says about Cavalier King Charles Spaniel

Computed from the 18,477 research dogs in the Atlas.

Dogs in the Atlas
102Founders
51 from Hayward2016, 34 from Momozawa, 10 from Spatola
Genetic diversity
0.24Tight
Mean heterozygosity across the breed. Ranks 11th most genetically tight of 107 ranked breeds.
What does genetic diversity mean?

How varied a breed's gene pool is — the share of gene spots where a typical dog of the breed carries two different versions rather than two identical ones.

How to read it: Higher = more diverse. Among well-sampled breeds it ranges roughly 0.22 (least diverse) to 0.33 (most diverse).

Diversity is a strength, not a verdict on any individual dog. Lower diversity means it's worth paying attention to recessive-risk testing — not that a dog is doomed.

Cluster structure
Splits into two genetic sub-populations
Intra-breed RMS distance: 26.75 · likely working/show-line, regional, or kennel lineage split.
What does within-breed variation mean?

How much individual dogs within the breed differ from each other genetically.

How to read it: Higher = more internal variety among individuals of the breed.

Sensitive to how many dogs of the breed we've sampled.

Related breeds
In the Toy group
Explore the full lineage map →
VBO foundation stock (breeding records) · AKC breed group
Relatedness is documented lineage + kennel family. Genetic-ancestry distance measures diversity, not kinship, so it isn't used here.
How long they live
11.8years (life expectancy)
95% CI 11.7–11.9 · VetCompass, McMillan 2024, n=13,782. source
What does typical lifespan mean?

The median age dogs of the breed tend to reach.

How to read it: Higher = longer-lived. Compare to longevity-for-size to see whether it's just a size effect.

Drawn from population lifespan records; individual dogs vary widely with care, genetics, and luck.

Trait genetics
Allele frequencies at named morphology loci

Frequency of the alternate allele in this breed at each locus's representative SNP.

Body size
IGF197%
HMGA272%
SMAD233%
LCORL100%
STC297%
ADAMTS1797%
Leg length
FGF4·CFA1872%
FGF4·CFA121%
Coat
RSPO228%
FGF532%
KRT7194%
MC1R29%
Ear set
MSRB397%
Skull shape
BMP370%
SMOC2100%
n = 102 dogs · high confidence · CanVAS (Brundage 2026) · Sniff Atlas
Names & origins

Other names

The Cavalier King Charles Spaniel is also recorded as CKCS.

Identified as Cavalier King Charles Spaniel (VBO:0200309) in the Vertebrate Breed Ontology (Mullen et al. 2025, CC-BY 4.0) · registry IDs FCI 136 · iDog 67 · VeNom 14844.

Temperament

What Cavalier King Charles Spaniels tend toward

Tendencies from owner surveys of purebred Cavalier King Charles Spaniels — a leaning across the breed, not a prediction for any one dog. A bar’s strength shows how much of that behavior breed actually explains: for most it’s faint, because the rest is your dog, their training, and the life you give them.

Proximity Seekingbreed ~13%
affectionatealoof
Environmental Engagementbreed ~9%
high engagementlow engagement
Biddabilitybreed ~18%
biddableindependent
Toy-directed Motor Patternsbreed ~18%
toy-directednot toy-directed
Agonistic Thresholdbreed ~9%
assertivediffident
Human Sociabilitybreed ~11%
less sociablehighly sociable
Arousal Levelbreed ~8%
arousedcomposed
Dog Sociabilitybreed ~8%
less sociablehighly sociable
n = 37 dogs · Morrill et al. 2022, Science, Darwin's Ark (CC0)
Owner-reported purebreds; each factor n ≥ 25. "Breed ~%" is the share of this behavior explained by breed.
What you see when you look at a Cavalier King Charles Spaniel

What does the genome say about how a Cavalier King Charles Spaniel looks?

Cavalier King Charles Spaniels look the way they do because of a small set of fixed and near-fixed morphology genes that, taken together, define the visible breed. Each translation below pairs the gene with the trait an owner actually sees, the breed's allele frequency at that locus, and a one-clause causal phrase.

Where the breed-defining genes act, mapped on a generic dog-body key — and how fixed each marker is in the Cavalier King Charles Spaniel. The figure is the most-settled marker we read in that region; the full per-locus panel is below. (The silhouette is a shared anatomical guide, not this breed's outline.)

Body sizeLCORL · 100%Skull shapeSMOC2 · 100%EarsMSRB3 · 97%Leg lengthFGF4 CFA18 · 72%Coat & colorKRT71 · 94%
CanVAS trait-locus panel (Brundage 2026)
15 morphology markers read across 5 regions. Allele frequency = how fixed a marker is in this breed, not whether your dog carries it.

Size and build

IGF1 is near-fixed at 97% for the small-body allele, which keeps the breed compact relative to its working-line ancestors.

IGF1what this gene does

IGF1 is a gene that plays a key role in determining a dog's body size. It influences how much a dog grows, affecting overall stature.

For your dog: Knowing about IGF1 gives you insight into your dog's size traits, but it’s just one part of the bigger picture when it comes to their health and care.

Full IGF1 gene page →

HMGA2 sits at 72%. HMGA2 is a chromosome-10 size locus that acts together with IGF1, and intermediate frequencies reflect partial commitment to the dominant size variant.

HMGA2what this gene does

HMGA2 is a gene that influences body size in dogs, helping determine how big or small a dog grows.

For your dog: Knowing about HMGA2 helps you appreciate the genetic factors behind your dog's size, but it doesn't signal any health issues.

Full HMGA2 gene page →

SMAD2 sits at 33% at the chromosome-7 height locus.

SMAD2what this gene does

SMAD2 is a gene involved in regulating body size by influencing how cells grow and develop.

For your dog: Knowing about SMAD2 helps understand your dog's size traits but isn't linked to health issues; no immediate action needed.

Full SMAD2 gene page →

LCORL is near-fixed at 100%, the NCAPG/LCORL height locus that is one of the strongest single contributors to canine body size.

LCORLwhat this gene does

LCORL is a gene that influences body size in dogs. It helps determine how big or small a dog might grow.

For your dog: Knowing about LCORL helps you appreciate the genetic factors behind your dog's size, but it’s just one piece of the bigger picture when it comes to health and care.

Full LCORL gene page →

STC2 is near-fixed at 97%, modulating growth-axis signaling toward the breed's body-size set point.

ADAMTS17 is at 97%, near-fixed for the size variant.

ADAMTS17what this gene does

ADAMTS17 is a gene that influences body size and also plays a role in certain eye conditions. It affects the structure of tissues in the eye and elsewhere in the body.

For your dog: If your dog belongs to a breed known to carry ADAMTS17 variants, it’s worth discussing genetic testing and eye exams with your vet to stay ahead of potential issues.

Full ADAMTS17 gene page →

Leg length

The FGF4 retrogene on chromosome 18 sits at 72%. This is the leg-length variant. The intermediate frequency means some dogs in this breed carry the short-legged allele and some do not.

The FGF4 retrogene on chromosome 12 is at 1%, leaving most of this breed clear of the chondrodystrophic intervertebral disc disease risk.

Coat type, length, and color

RSPO2 is at 28% for the furnishings allele. The breed does not carry the eyebrows-and-mustache pattern of Wheatens, Schnauzers, or wire-haired terriers.

RSPO2what this gene does

RSPO2 influences the texture and appearance of a dog's coat, particularly the presence of 'furnishings' like mustaches and eyebrows. It helps determine whether a dog has that distinctive wiry or textured look.

For your dog: If your dog has those wiry eyebrows or a mustache, RSPO2 is part of the reason—no health worries, just a coat feature worth knowing about.

Full RSPO2 gene page →

FGF5 sits at 32% for the long-coat variant. Coat length is influenced by other loci as well, so intermediate FGF5 frequencies do not always correspond to intermediate visible coat lengths.

FGF5what this gene does

FGF5 is a gene that influences the length of a dog's coat. It acts like a natural switch, telling hair follicles when to stop growing longer fur.

For your dog: If your dog has a notably long or short coat, FGF5 is likely part of the reason—no action needed, but it’s a neat genetic detail to know.

Full FGF5 gene page →

KRT71 is near-fixed at 94% for the wavy/curly variant. Coat curl phenotype varies across breeds at this fixation depending on modifier loci, and visible expression is not always curled even when the locus is fixed.

KRT71what this gene does

KRT71 is a gene that influences the curliness of a dog's coat. It helps determine whether a dog's fur is straight or has a distinctive curl.

For your dog: If your dog has a curly coat, KRT71 is likely part of the reason; it’s a natural variation, not a health concern.

Full KRT71 gene page →

MC1R is at 29% at the representative SNP, leaving the breed in the black-to-brown coat range under the dominant E allele.

MC1Rwhat this gene does

MC1R is a gene that influences coat color in dogs, affecting how pigments are produced in the fur.

For your dog: Knowing about MC1R gives insight into your dog's coat color but doesn't relate to health issues.

Full MC1R gene page →

Ears

MSRB3 is at 97% for the drop-ear allele, the genetic basis of the breed's signature dropped ear set.

MSRB3what this gene does

MSRB3 is a gene involved in the development of ear shape and structure in dogs.

For your dog: Understanding MSRB3 helps explain why your dog's ears look the way they do, but it isn't linked to any health issues.

Full MSRB3 gene page →

Skull shape

BMP3 sits at 70%, contributing to the breed's moderate, mesaticephalic head shape rather than the extreme brachycephalic form.

BMP3what this gene does

BMP3 is a gene that influences the shape of a dog's skull, particularly contributing to a shorter, broader head shape known as brachycephaly.

For your dog: If your dog has a broad, short skull, it's worth discussing with your vet how this might impact their health, even though BMP3 isn't directly tied to illness.

Full BMP3 gene page →

SMOC2 is at 100%, the major locus contributing to the breed's brachycephalic face shape.

SMOC2what this gene does

SMOC2 influences the shape of a dog's skull, particularly affecting how flat or short the face appears.

For your dog: If your dog has a short nose, it's worth discussing with your vet how this trait might impact their health over time.

Full SMOC2 gene page →
Mendelian-disease genetics

What genetic diseases do Cavalier King Charles Spaniels carry?

From a panel of 250 Mendelian-disease variants screened in 1,054,293 dogs (Donner et al. 2023), Cavalier King Charles Spaniels carry 14 of them at observable frequency. Carrier frequency is not clinical risk. Most recessive variants require two copies for disease expression; many dominant variants show incomplete penetrance. Read this as a population fingerprint of what's in the gene pool, not a per-dog prediction.

n = 2,232 dogs · 1 variant tested · OMIA:000157-9615 · omia.org →
FGF4what this gene does

FGF4 influences leg length by affecting bone growth, leading to shorter legs in certain breeds.

For your dog: If your dog is from a breed known to carry this gene, it's worth discussing spinal health with your vet, but being a carrier doesn’t guarantee problems.

Degenerative Myelopathy (DM)
Autosomal recessive (Incomplete penetrance)
high 51.0%
n = 2,242 dogs · 1 variant tested · OMIA:000263-9615 · omia.org →
SOD1what this gene does

SOD1 is a gene that helps protect cells from damage caused by harmful molecules called free radicals.

For your dog: If your dog is a carrier of SOD1 variants, it's worth discussing with your vet, but remember carrier status doesn't mean your dog will get the disease.

Episodic Falling (EF)
Autosomal recessive
low 6.7%
n = 2,243 dogs · 1 variant tested · OMIA:001592-9615 · omia.org →
Skeletal Dysplasia 2 (SD2)
Autosomal recessive
low 1.5%
n = 2,243 dogs · 1 variant tested · OMIA:001772-9615 · omia.org →
COL11A2what this gene does

COL11A2 is a gene that helps produce a type of collagen important for healthy bones and cartilage.

For your dog: If your dog is from a breed known to carry COL11A2 variants, it's worth discussing genetic testing with your vet to understand any risks.

n = 2,243 dogs · 1 variant tested · OMIA:001298-9615 · omia.org →
PRCDwhat this gene does

PRCD is a gene involved in the health of a dog's retina, the part of the eye that detects light and helps with vision.

For your dog: If your dog belongs to a breed known to carry PRCD changes, it's worth discussing eye health and potential genetic testing with your vet.

Prekallikrein Deficiency
Autosomal recessive
low 0.13%
n = 2,243 dogs · 1 variant tested · OMIA:000819-9615 · omia.org →
KLKB1what this gene does

KLKB1 is a gene that helps produce prekallikrein, a protein involved in blood clotting and inflammation.

For your dog: If your dog is from a breed known to carry KLKB1 variants, it's worth asking your vet about blood clotting tests, especially before surgeries.

Cone-Rod Dystrophy (cord1-PRA/crd4)
Autosomal recessive (Incomplete penetrance)
low <0.1%
n = 2,242 dogs · 1 variant tested · OMIA:001432-9615 · omia.org →
RPGRIP1what this gene does

RPGRIP1 is a gene involved in the function of photoreceptor cells in the eye, which help dogs see in different light conditions.

For your dog: If your dog belongs to a breed known to carry RPGRIP1 mutations, it’s worth discussing with your vet to understand the risks and monitor eye health.

Exercise-Induced Collapse (EIC)
Autosomal recessive (Incomplete penetrance)
low <0.1%
n = 2,242 dogs · 1 variant tested · OMIA:001466-9615 · omia.org →
DNM1what this gene does

DNM1 is a gene that helps nerve cells communicate properly by managing how they send signals during muscle activity.

For your dog: If your dog belongs to one of the breeds known to carry this gene variant, it's worth discussing EIC with your vet, especially if your dog is very active or shows signs of weakness during exercise.

Factor VII Deficiency
Autosomal recessive
low <0.1%
n = 2,243 dogs · 1 variant tested · OMIA:000361-9615 · omia.org →
F7what this gene does

The F7 gene helps produce a protein important for blood clotting, which stops bleeding when dogs get injured.

For your dog: If your dog is from a breed known to carry F7 variants, it's worth mentioning to your vet before any surgery or if you notice unusual bleeding.

low <0.1%
n = 2,243 dogs · 1 variant tested · OMIA:001514-9615 · omia.org →
GDNFwhat this gene does

GDNF is a gene that helps support nerve cells, especially those involved in sensing pain and movement.

For your dog: If your dog is from a breed that can carry this gene change, it’s worth asking your vet about testing to understand any potential risks.

n = 2,243 dogs · 1 variant tested · OMIA:001400-9615 · omia.org →
SLC13A1what this gene does

SLC13A1 is a gene that helps transport important molecules involved in bone and cartilage health. It plays a role in maintaining the structure and function of these tissues.

For your dog: If your dog is one of the breeds known to carry this gene variant, it’s worth discussing with your vet to understand what it means for their bone health and care.

Primary Lens Luxation (PLL)
Autosomal recessive
low <0.1%
n = 2,243 dogs · 2 variants tested · OMIA:000588-9615 · omia.org →
ADAMTS17what this gene does

ADAMTS17 is a gene that influences body size and also plays a role in certain eye conditions. It affects the structure of tissues in the eye and elsewhere in the body.

For your dog: If your dog belongs to a breed known to carry ADAMTS17 variants, it’s worth discussing genetic testing and eye exams with your vet to stay ahead of potential issues.

Full ADAMTS17 gene page →
Source: Donner J et al. 2023. Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651 · Evidence: Limited (DTC ascertainment, tag-SNP proxy) · Confounding MEDIUM · License CC-BY-4.0 · Phene IDs from OMIA (Sydney School of Veterinary Science, The University of Sydney; DOI 10.25910/2AMR-PV70).
Sample size in this breed: 2,243 dogs from the Donner 2023 cohort.

Which Mendelian variants matter most for Cavalier King Charles Spaniels?

The Mendelian-disease table above lists variants screened in 2,243 Cavaliers (Donner 2023). Two dominate by frequency and clinical impact. A third shapes breeding decisions. The rest are rare.

Chondrodystrophy and Intervertebral Disc Disease Risk (CDDY)

Chondrodystrophy and Intervertebral Disc Disease Risk in Cavaliers is caused by a dominant FGF4 retrogene insertion. Cavaliers carry the CDDY variant at 99% (Donner 2023, n=2,232), meaning nearly every Cavalier in existence carries at least one copy.

Here is what makes this case unusual. Dachshunds, Corgis, and Basset Hounds with this variant show shortened limbs and a distinctive chondrodystrophic body shape. Cavaliers carry the same genotype but do not. The breed standard shows normal leg proportions. In Cavaliers, the relevant clinical expression of this variant is elevated intervertebral disc disease risk: disc herniation, spinal cord compression, and hind-limb weakness in middle age.

The 99% frequency means testing for carrier status is not useful for breeding decisions. Every breeding pair will carry at least one copy. Instead, the clinical relevance is management: Cavalier owners should understand that IVDD is not a rare or surprising diagnosis in the breed. It is a breed-typical risk. Weight management, controlled exercise, and awareness of first-sign symptoms (back pain, reluctance to jump, hind-limb weakness) are the practical takeaways.

Degenerative Myelopathy (DM)

Degenerative Myelopathy in Cavaliers is a progressive spinal-cord degeneration caused by a recessive variant with incomplete penetrance. Affected dogs typically begin showing hind-limb weakness and loss of coordination in middle age, progressing to paralysis over months to years. Roughly half of Cavaliers are carriers (51.0%, Donner 2023, n=2,242), meaning one in four Cavalier-to-Cavalier pairings will produce at-risk offspring.

Not every dog with two copies develops symptoms, which is why the inheritance is incomplete penetrance. The Donner 2023 cohort’s phenotype-confirmation data support real incomplete penetrance: carriers in the screened population outnumber phenotype-confirmed affected dogs. Testing is available and useful for breeding stock planning.

Episodic Falling (EF)

Episodic Falling in Cavaliers is a rare autosomal-recessive condition characterized by sudden collapse episodes triggered by exercise or excitement. Affected dogs lose muscle control briefly, then recover. It is distressing to witness but not life-threatening. The variant is rare in the breed: 6.7% carrier frequency (Donner 2023, n=2,243). No at-risk dogs in the Donner cohort were phenotype-confirmed, suggesting low penetrance or rarity in practice.

How should I test my Cavalier?

A breed-specific panel is the efficient path. The minimum useful set for Cavaliers is CDDY (intervertebral disc disease risk), DM (degenerative myelopathy), and episodic falling. Testing is available from CLIA-accredited labs including the PennGen Laboratory. The Cavalier King Charles Spaniel Club USA maintains health screening recommendations for breeding stock.

What should I feed a Cavalier King Charles Spaniel?

Feeding a Cavalier well means feeding around two breed-defining vulnerabilities: mitral valve disease that emerges in middle age, and the near-ubiquitous IVDD variant (99% carrier frequency, Donner 2023, n=2,232) that shapes spinal-cord risk across the lifespan.

Cardiac health starts with sodium and taurine awareness. Mitral valve disease (MVD) affects a large fraction of Cavaliers by middle age, with roughly half developing a murmur by age five (Häggström et al. 1992, J Small Anim Pract 33:253-258). Breed-club screening programs track murmur progression from early detection through symptomatic disease. Once a murmur is present, dietary sodium restriction becomes part of the medical plan. Before a murmur appears, taurine sufficiency is the guardrail. Grain-free diets have been associated with taurine depletion and dilated cardiomyopathy in several breeds (Adin et al. 2019, JVIM, DOI:10.1111/jvim.15402). For Cavaliers with emerging cardiac disease, a complete, balanced, grain-inclusive adult formula is the safer default.

Joint and spine care is a daily consideration. The IVDD variant is fixed in the breed. Weight management is not optional; it is foundational spine protection. A Cavalier at ideal body condition reduces disc herniation risk compared to an overweight peer. Puppy formulations should use the NRC 2006 calcium-to-phosphorus guideline of 1.1:1 to 2:1 to avoid skeletal overgrowth stress. Adult maintenance formulations should support lean body mass without excess calories. Some breeders and veterinarians add joint-support supplements (glucosamine, chondroitin) as preventive; peer-reviewed evidence for efficacy is mixed, but harm is minimal if renal function is normal.

Feed small, frequent meals as the dog ages. Cavaliers are toy-breed sized, and small, frequent meals support steady energy levels across the day. Older dogs benefit from two or three small meals rather than one large one, reducing gastric distension and supporting steady glucose availability through the day.

What we don’t know

We do not yet understand why the 99% CDDY carrier frequency in Cavaliers does not produce the classical chondrodystrophic phenotype that fixed populations of Dachshunds and Corgis show. The genotype is identical; the phenotype is breed-modified. The mechanism of that modification is unknown.

The penetrance of degenerative myelopathy in Cavaliers is incompletely characterized. We know roughly half the breed carries the variant and that not all carriers phenotype. We do not know which environmental, genetic, or polygenic factors determine which carriers become symptomatic or at what age. The published environmental analyses have come back mostly null.

Mitral valve disease prevalence and onset timing in Cavaliers have been studied in detail by cardiology researchers, but the genetic architecture beyond the known common variants remains unsettled. Multiple loci likely contribute, and the interaction between genotype, age, sex, and body size is incompletely modeled.

Frequently asked questions about Cavalier King Charles Spaniels

What is the most common health problem in Cavaliers? Mitral valve disease. Published cardiac screening data show roughly half of Cavaliers develop a murmur by age five (Häggström et al. 1992, J Small Anim Pract 33:253-258). It is the breed-typical condition that shapes veterinary care across the lifespan.

Are Cavaliers prone to back problems? Yes. Cavaliers carry the CDDY variant at 99% (Donner 2023, n=2,232). Intervertebral disc disease is the breed-typical expression. Weight management and controlled exercise reduce risk; awareness of first symptoms (back pain, reluctance to jump, hind-limb weakness) is essential.

How long do Cavaliers live? The atlas-derived median lifespan for Cavaliers is 10.0 years. Individual dogs vary widely based on genetics, cardiac status, and management. Regular cardiac screening and weight management extend healthy years.

Should I do a DNA test on my Cavalier? For breeding stock, yes. For pet dogs, testing clarifies degenerative myelopathy and episodic falling risk, which can inform exercise and management decisions. The CDDY variant is fixed (99%) so testing for carrier status is not useful.

What is the best diet for a Cavalier? A complete, balanced, grain-inclusive adult formula is the safest default, especially as the dog ages and cardiac screening becomes routine. Once a murmur is detected, sodium restriction becomes part of the medical plan. Avoid pulse-heavy, grain-free diets in this breed.

Are Cavaliers good with kids? Yes. Cavaliers were bred for companionship in royal courts and remain deeply people-oriented. They are typically gentle and patient with children. Individual temperament varies; early socialization is always important.

Can Cavaliers handle hot weather? Cavaliers are not brachycephalic (flat-faced), so heat tolerance is better than in Bulldogs or Pugs. They still benefit from shade, fresh water, and rest during hot hours. Exercise should be timed for cooler parts of the day in summer.

What is degenerative myelopathy and should I worry about it? Degenerative myelopathy is a progressive spinal-cord disease caused by a recessive variant with incomplete penetrance. About 51% of Cavaliers carry the variant (Donner 2023, n=2,242), but not all carriers develop symptoms. Testing is available and can inform breeding decisions and management planning.

A gift to human medicine

Cavalier King Charles Spaniels are a natural model for human disease

Because the same genes cause the same conditions across species, the inherited conditions documented in Cavalier King Charles Spaniels help researchers understand, and work toward treating, the human diseases they model. This is the dog advancing human medicine. The breed models the human disease; it does not have it, and this is not a prediction for your dog.

Human equivalents via OMIA → Mondo / OMIM. Model-of, not identity.
Documented in OMIA

Every condition recorded in the Cavalier King Charles Spaniel

Beyond the testable carriers above, OMIA's literature catalogue records 18 genetic conditions in the Cavalier King Charles Spaniel, 16 of which have a known human equivalent. This is the documented landscape across all Cavalier King Charles Spaniels ever studied, not a prediction for any one dog.

Online Mendelian Inheritance in Animals (OMIA); Nicholas, Tammen & Sydney Informatics Hub, DOI 10.25910/2AMR-PV70
Documented in the breed's literature is not carrier status and not a forecast for an individual dog. Human equivalents are mapped via Mondo/OMIM. Carrier frequencies (above) are the separately-measured testable subset (Donner 2023).
The data behind this page

Where every number on this page came from.

This page draws on three primary data sources. Carrier frequencies for the Mendelian section come from Donner et al. 2023 (CC-BY-4.0). We grade these data at evidence Limited because the cohort is a direct-to-consumer ascertainment, which biases toward owners who chose to test their dogs. The panel also uses tag-SNP proxies for some variants rather than direct causal-variant assays. Limited is a study-design grade, not a quality grade: the Donner cohort is the largest open canine-genotype dataset in existence and we are grateful for it. We rate the confounding MEDIUM.

Population-genetic dimensions (heterozygosity, intra-breed PCA distance, nearest neighbors, trait-locus frequencies) come from CanVAS (Brundage 2026), harmonized through the Sniff Atlas. The exact release date and verification commit are pinned at the bottom of the page so a researcher can trace a number back to a specific snapshot. The disease-gene-variant graph comes from OMIA (Online Mendelian Inheritance in Animals; Nicholas, Tammen, and the Sydney Informatics Hub at the Sydney School of Veterinary Science, The University of Sydney; retrieved April 2026, DOI 10.25910/2AMR-PV70).

What this page does not yet have. Inheritance modes and per-disease penetrance evidence from Donner 2023 are now in the structured data for every variant the panel covers. Mondo, OMIM, Ensembl, and HGNC cross-references on gene pages remain pending, they arrive in December 2026 alongside the imputed 9.67M-variant CanVAS dataset via the OMIA SQL dump absorption. Until then, gene IDs carry NCBI Gene and OMIA phene URLs only; the wider human-homolog and disease-ontology cross-reference set fills in with that release.

How to cite this page. The computed dimensions on this page are derived from the open Sniff Atlas v1.0.1 (Gehring 2026, doi:10.5281/zenodo.20566358, CC-BY 4.0). Full citation formats including BibTeX, RIS, and CITATION.cff at sniff.world/cite.

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References
  1. Donner J, Freyer J, Davison S, Anderson H, Blades M, Honkanen L, et al. (2023). Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651. doi:10.1371/journal.pgen.1010651
  2. Brundage J, et al. (2026). CanVAS: a harmonized canine variant atlas. bioRxiv. doi:10.64898/2026.04.13.718238
  3. Nicholas, F.W., Tammen, I., & Sydney Informatics Hub. (2026). Online Mendelian Inheritance in Animals (OMIA) [dataset]. The University of Sydney. https://omia.org. doi:10.25910/2AMR-PV70 (retrieved April 2026).
Last updated
Sources: CanVAS (Brundage 2026) · Donner 2023 · OMIA