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Dachshund

Dachshund
Photo: Katemil94 / CC BY-SA 4.0 · Wikimedia

97 Dachshunds in the atlas. Every number on this page has a source.

Population-genetic snapshot of Dachshunds in the Sniff Atlas, source-graded Mendelian carrier frequencies from Donner 2023, and nutrition guidance tied to the genetic findings above.

Also known as Bassotto, Dackel, Jamnik, and Perro Salchicha.

The plain version

Dachshunds have a moderately diverse genetic background. They’re small dogs, usually weighing around 24 pounds, and tend to live about 13 to 14 years. Their unique long body and short legs make them easy to recognize and great for cozy homes. No specific genetic health concerns were found in this breed’s gene pool, but regular vet check-ups are always a good idea.

What the atlas says about Dachshund

In the atlas, the Dachshund clusters consistently as Dachshund (100% of the 97 dogs here). Genetic diversity is high (mean heterozygosity 0.3282), reflecting either a mixed-breed cluster or breeds with broad genetic backgrounds. At the trait loci, FGF4_retrogene_CFA18 runs lower than average (0% here vs 77%); FGF4_retrogene_CFA12 runs lower than average (18% here vs 80%).

Mean heterozygosity is 0.328, notably high, indicates broad genetic background.

Median lifespan is 13.6 years, slightly longer than expected for the breed size (10.9 kg).

Genetic dimensions · CanVAS atlas

What the genome says about Dachshund

Computed from the 18,477 research dogs in the Atlas.

Dogs in the Atlas
97Founders
40 from Momozawa, 28 from Hayward2016, 12 from Shannon
Genetic diversity
0.33Moderate
Mean heterozygosity across the breed. Ranks 74th most genetically tight of 107 ranked breeds.
What does genetic diversity mean?

How varied a breed's gene pool is — the share of gene spots where a typical dog of the breed carries two different versions rather than two identical ones.

How to read it: Higher = more diverse. Among well-sampled breeds it ranges roughly 0.22 (least diverse) to 0.33 (most diverse).

Diversity is a strength, not a verdict on any individual dog. Lower diversity means it's worth paying attention to recessive-risk testing — not that a dog is doomed.

Cluster structure
Splits into two genetic sub-populations
Intra-breed RMS distance: 28.20 · likely working/show-line, regional, or kennel lineage split.
What does within-breed variation mean?

How much individual dogs within the breed differ from each other genetically.

How to read it: Higher = more internal variety among individuals of the breed.

Sensitive to how many dogs of the breed we've sampled.

Related breeds
Gave rise to
In the Hound group
Explore the full lineage map →
VBO foundation stock (breeding records) · AKC breed group
Relatedness is documented lineage + kennel family. Genetic-ancestry distance measures diversity, not kinship, so it isn't used here.
How long they live
13.2years (life expectancy)
95% CI 13–13.5 · VetCompass, McMillan 2024, n=2,305. source
What does typical lifespan mean?

The median age dogs of the breed tend to reach.

How to read it: Higher = longer-lived. Compare to longevity-for-size to see whether it's just a size effect.

Drawn from population lifespan records; individual dogs vary widely with care, genetics, and luck.

Trait genetics
Allele frequencies at named morphology loci

Frequency of the alternate allele in this breed at each locus's representative SNP.

Body size
IGF176%
HMGA283%
SMAD226%
LCORL95%
STC262%
ADAMTS1781%
Leg length
FGF4·CFA181%
FGF4·CFA1218%
Coat
RSPO281%
FGF580%
KRT7186%
MC1R92%
Ear set
MSRB393%
Skull shape
BMP387%
SMOC293%
n = 97 dogs · high confidence · CanVAS (Brundage 2026) · Sniff Atlas
Names & origins

Other names

The Dachshund is also recorded as Bassotto, Dackel, Jamnik, Perro Salchicha, Salsichinha, Sausage Dog, Taksis, and Tax.

Identified as Dachshund (VBO:0200406) in the Vertebrate Breed Ontology (Mullen et al. 2025, CC-BY 4.0) · registry IDs FCI 148 · iDog 83 · VeNom 13952.

Temperament

What Dachshunds tend toward

Tendencies from owner surveys of purebred Dachshunds — a leaning across the breed, not a prediction for any one dog. A bar’s strength shows how much of that behavior breed actually explains: for most it’s faint, because the rest is your dog, their training, and the life you give them.

Biddabilitybreed ~18%
biddableindependent
Dog Sociabilitybreed ~8%
less sociablehighly sociable
Proximity Seekingbreed ~13%
affectionatealoof
Environmental Engagementbreed ~9%
high engagementlow engagement
Arousal Levelbreed ~8%
arousedcomposed
Human Sociabilitybreed ~11%
less sociablehighly sociable
Agonistic Thresholdbreed ~9%
assertivediffident
Toy-directed Motor Patternsbreed ~18%
toy-directednot toy-directed
n = 68 dogs · Morrill et al. 2022, Science, Darwin's Ark (CC0)
Owner-reported purebreds; each factor n ≥ 25. "Breed ~%" is the share of this behavior explained by breed.
What you see when you look at a Dachshund

What does the genome say about how a Dachshund looks?

Dachshunds look the way they do because of a small set of fixed and near-fixed morphology genes that, taken together, define the visible breed. Each translation below pairs the gene with the trait an owner actually sees, the breed's allele frequency at that locus, and a one-clause causal phrase.

Where the breed-defining genes act, mapped on a generic dog-body key — and how fixed each marker is in the Dachshund. The figure is the most-settled marker we read in that region; the full per-locus panel is below. (The silhouette is a shared anatomical guide, not this breed's outline.)

Body sizeLCORL · 95%Skull shapeSMOC2 · 93%EarsMSRB3 · 93%Leg lengthFGF4 CFA12 · 18%Coat & colorMC1R · 92%
CanVAS trait-locus panel (Brundage 2026)
15 morphology markers read across 5 regions. Allele frequency = how fixed a marker is in this breed, not whether your dog carries it.

Size and build

IGF1 sits at 76% for the small-body allele. IGF1 is the gene that sets dog body size from Chihuahua to Great Dane. Intermediate frequencies typically keep a breed in the mid-sized range rather than tipping toward the larger working forms.

IGF1what this gene does

IGF1 is a gene that plays a key role in determining a dog's body size. It influences how much a dog grows, affecting overall stature.

For your dog: Knowing about IGF1 gives you insight into your dog's size traits, but it’s just one part of the bigger picture when it comes to their health and care.

Full IGF1 gene page →

HMGA2 sits at 83%. HMGA2 is a chromosome-10 size locus that acts together with IGF1, and intermediate frequencies reflect partial commitment to the dominant size variant.

HMGA2what this gene does

HMGA2 is a gene that influences body size in dogs, helping determine how big or small a dog grows.

For your dog: Knowing about HMGA2 helps you appreciate the genetic factors behind your dog's size, but it doesn't signal any health issues.

Full HMGA2 gene page →

SMAD2 is at 26%, leaving the height signal mostly to other size genes.

SMAD2what this gene does

SMAD2 is a gene involved in regulating body size by influencing how cells grow and develop.

For your dog: Knowing about SMAD2 helps understand your dog's size traits but isn't linked to health issues; no immediate action needed.

Full SMAD2 gene page →

LCORL is near-fixed at 95%, the NCAPG/LCORL height locus that is one of the strongest single contributors to canine body size.

LCORLwhat this gene does

LCORL is a gene that influences body size in dogs. It helps determine how big or small a dog might grow.

For your dog: Knowing about LCORL helps you appreciate the genetic factors behind your dog's size, but it’s just one piece of the bigger picture when it comes to health and care.

Full LCORL gene page →

STC2 sits at 62%.

ADAMTS17 sits at 81%. ADAMTS17 is a body-size locus also linked to lens disorders.

ADAMTS17what this gene does

ADAMTS17 is a gene that influences body size and also plays a role in certain eye conditions. It affects the structure of tissues in the eye and elsewhere in the body.

For your dog: If your dog belongs to a breed known to carry ADAMTS17 variants, it’s worth discussing genetic testing and eye exams with your vet to stay ahead of potential issues.

Full ADAMTS17 gene page →

Leg length

The FGF4 retrogene on chromosome 18 is at 1%, the chromosome-18 leg-length variant, which keeps the breed short-legged like Corgis and Dachshunds.

The FGF4 retrogene on chromosome 12 is at 18%, leaving most of this breed clear of the chondrodystrophic intervertebral disc disease risk.

Coat type, length, and color

RSPO2 sits at 81% for the furnishings variant. Furnishings (the eyebrow-and-mustache pattern seen in Schnauzers and Wheaten Terriers) vary across the population at this intermediate frequency, and visible expression depends on the specific allele combination each dog carries.

RSPO2what this gene does

RSPO2 influences the texture and appearance of a dog's coat, particularly the presence of 'furnishings' like mustaches and eyebrows. It helps determine whether a dog has that distinctive wiry or textured look.

For your dog: If your dog has those wiry eyebrows or a mustache, RSPO2 is part of the reason—no health worries, just a coat feature worth knowing about.

Full RSPO2 gene page →

FGF5 sits at 80% for the long-coat variant. Coat length is influenced by other loci as well, so intermediate FGF5 frequencies do not always correspond to intermediate visible coat lengths.

FGF5what this gene does

FGF5 is a gene that influences the length of a dog's coat. It acts like a natural switch, telling hair follicles when to stop growing longer fur.

For your dog: If your dog has a notably long or short coat, FGF5 is likely part of the reason—no action needed, but it’s a neat genetic detail to know.

Full FGF5 gene page →

KRT71 is near-fixed at 86% for the wavy/curly variant. Coat curl phenotype varies across breeds at this fixation depending on modifier loci, and visible expression is not always curled even when the locus is fixed.

KRT71what this gene does

KRT71 is a gene that influences the curliness of a dog's coat. It helps determine whether a dog's fur is straight or has a distinctive curl.

For your dog: If your dog has a curly coat, KRT71 is likely part of the reason; it’s a natural variation, not a health concern.

Full KRT71 gene page →

MC1R is at 92% at the representative SNP. MC1R controls the switch between red-to-gold and black-to-brown pigment, with the e/e homozygous genotype producing the gold-to-red spectrum by blocking eumelanin (black and brown pigment).

MC1Rwhat this gene does

MC1R is a gene that influences coat color in dogs, affecting how pigments are produced in the fur.

For your dog: Knowing about MC1R gives insight into your dog's coat color but doesn't relate to health issues.

Full MC1R gene page →

Ears

MSRB3 is at 93% for the drop-ear allele, the genetic basis of the breed's signature dropped ear set.

MSRB3what this gene does

MSRB3 is a gene involved in the development of ear shape and structure in dogs.

For your dog: Understanding MSRB3 helps explain why your dog's ears look the way they do, but it isn't linked to any health issues.

Full MSRB3 gene page →

Skull shape

BMP3 is at 87%, contributing to the breed's brachycephalic skull shape.

BMP3what this gene does

BMP3 is a gene that influences the shape of a dog's skull, particularly contributing to a shorter, broader head shape known as brachycephaly.

For your dog: If your dog has a broad, short skull, it's worth discussing with your vet how this might impact their health, even though BMP3 isn't directly tied to illness.

Full BMP3 gene page →

SMOC2 is at 93%, the major locus contributing to the breed's brachycephalic face shape.

SMOC2what this gene does

SMOC2 influences the shape of a dog's skull, particularly affecting how flat or short the face appears.

For your dog: If your dog has a short nose, it's worth discussing with your vet how this trait might impact their health over time.

Full SMOC2 gene page →
Mendelian-disease genetics

What genetic diseases do Dachshunds carry?

From a panel of 250 Mendelian-disease variants screened in 1,054,293 dogs (Donner et al. 2023), Dachshunds carry 14 of them at observable frequency. Carrier frequency is not clinical risk. Most recessive variants require two copies for disease expression; many dominant variants show incomplete penetrance. Read this as a population fingerprint of what's in the gene pool, not a per-dog prediction.

n = 6 dogs · 1 variant tested · OMIA:000157-9615 · omia.org →
FGF4what this gene does

FGF4 influences leg length by affecting bone growth, leading to shorter legs in certain breeds.

For your dog: If your dog is from a breed known to carry this gene, it's worth discussing spinal health with your vet, but being a carrier doesn’t guarantee problems.

Cone-Rod Dystrophy (cord1-PRA/crd4)
Autosomal recessive (Incomplete penetrance)
high 34.7%
n = 213 dogs · 1 variant tested · OMIA:001432-9615 · omia.org →
RPGRIP1what this gene does

RPGRIP1 is a gene involved in the function of photoreceptor cells in the eye, which help dogs see in different light conditions.

For your dog: If your dog belongs to a breed known to carry RPGRIP1 mutations, it’s worth discussing with your vet to understand the risks and monitor eye health.

n = 585 dogs · 1 variant tested · OMIA:001483-9615 · omia.org →
Degenerative Myelopathy (DM)
Autosomal recessive (Incomplete penetrance)
low 0.60%
n = 585 dogs · 1 variant tested · OMIA:000263-9615 · omia.org →
SOD1what this gene does

SOD1 is a gene that helps protect cells from damage caused by harmful molecules called free radicals.

For your dog: If your dog is a carrier of SOD1 variants, it's worth discussing with your vet, but remember carrier status doesn't mean your dog will get the disease.

low 0.23%
n = 213 dogs · 1 variant tested · OMIA:001514-9615 · omia.org →
GDNFwhat this gene does

GDNF is a gene that helps support nerve cells, especially those involved in sensing pain and movement.

For your dog: If your dog is from a breed that can carry this gene change, it’s worth asking your vet about testing to understand any potential risks.

n = 213 dogs · 2 variants tested · OMIA:000162-9615 · omia.org →
PDK4what this gene does

PDK4 helps regulate how cells use energy, especially in the heart muscle.

For your dog: If your dog is one of the breeds known to carry this gene, it’s worth discussing heart health with your vet, but being a carrier doesn’t mean your dog will develop disease.

n = 579 dogs · 1 variant tested · OMIA:001970-9615 · omia.org →
RAB3GAP1what this gene does

RAB3GAP1 is a gene involved in nerve cell function, particularly in how cells communicate and maintain their structure.

For your dog: If your dog is one of the breeds known to carry this gene variant, it's worth discussing genetic testing with your vet to understand any potential risks.

Factor VII Deficiency
Autosomal recessive
low <0.1%
n = 585 dogs · 1 variant tested · OMIA:000361-9615 · omia.org →
F7what this gene does

The F7 gene helps produce a protein important for blood clotting, which stops bleeding when dogs get injured.

For your dog: If your dog is from a breed known to carry F7 variants, it's worth mentioning to your vet before any surgery or if you notice unusual bleeding.

Leonberger Polyneuropathy, Type 2 (LPN2)
Autosomal dominant (Incomplete penetrance)
low <0.1%
n = 585 dogs · 1 variant tested · OMIA:002119-9615 · omia.org →
Primary Lens Luxation (PLL)
Autosomal recessive
low <0.1%
n = 585 dogs · 2 variants tested · OMIA:000588-9615 · omia.org →
ADAMTS17what this gene does

ADAMTS17 is a gene that influences body size and also plays a role in certain eye conditions. It affects the structure of tissues in the eye and elsewhere in the body.

For your dog: If your dog belongs to a breed known to carry ADAMTS17 variants, it’s worth discussing genetic testing and eye exams with your vet to stay ahead of potential issues.

Full ADAMTS17 gene page →
n = 585 dogs · 1 variant tested · OMIA:001298-9615 · omia.org →
PRCDwhat this gene does

PRCD is a gene involved in the health of a dog's retina, the part of the eye that detects light and helps with vision.

For your dog: If your dog belongs to a breed known to carry PRCD changes, it's worth discussing eye health and potential genetic testing with your vet.

low <0.1%
n = 585 dogs · 1 variant tested · OMIA:001057-9615 · omia.org →
Source: Donner J et al. 2023. Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651 · Evidence: Limited (DTC ascertainment, tag-SNP proxy) · Confounding MEDIUM · License CC-BY-4.0 · Phene IDs from OMIA (Sydney School of Veterinary Science, The University of Sydney; DOI 10.25910/2AMR-PV70).
Sample size in this breed: 809 dogs from the Donner 2023 cohort.

Which Mendelian variants matter most for Dachshunds?

The Donner 2023 cohort did not identify Mendelian variants at observable carrier frequency in Dachshunds. That absence of a long list is unusual and meaningful. It does not mean Dachshunds are genetically simple. It means the breed’s known heritable architecture, the traits that make a Dachshund a Dachshund, traces most clearly to morphology genes rather than to recessive disease variants found at clinical frequency.

The FGF4 retrogene on chromosome 12 (CFA12) is present at 18% allele frequency in the breed; a second FGF4 retrogene on chromosome 18 (CFA18) is present at 1%. This variant drives chondrodystrophic dwarfism (CDDY), which includes shortened limbs and predisposition to intervertebral disc disease (IVDD), the spinal condition Dachshunds are most associated with. The variant does not cause IVDD in every carrier; penetrance is incomplete and modulated by age, weight, activity, and spine anatomy. But the 18% frequency means IVDD risk is built into the breed’s genetic architecture in a way it is not for most other dogs.

Dachshunds do not show the classical chondrodystrophic limb phenotype as uniformly as they once did, especially in show lines trending toward longer legs. The morphology gene allele frequencies show variation in FGF4 retrogene load (CFA12 18%, CFA18 1%), size genes (IGF1 76%, HMGA2 83%), and leg-length modifiers (STC2 62%, ADAMTS17 81%). That variation reflects decades of breeding decisions within the breed. The IVDD risk remains. The shortened-limb phenotype varies across lines, with some show-line dogs trending toward relatively longer legs.

How should I test my Dachshund?

A Dachshund does not need a Mendelian-disease panel because the breed has not shown publishable Mendelian variants at clinical frequency. That said, spinal health matters more for this breed than for almost any other. Work with your vet to keep your Dachshund at a lean weight, avoid high-impact jumping from furniture, and, if your Dachshund shows signs of hind-limb weakness, reluctance to jump, or back pain, get imaging promptly. Early imaging can catch disc bulging before acute extrusion. An MRI is the gold standard; a CT is a practical alternative if MRI is not available.

What should I feed a Dachshund?

Feeding a Dachshund well means managing the breed’s intervertebral disc disease risk through weight control and joint support, because spinal health is non-negotiable in a dog built this low to the ground.

Weight management is the single most important nutritional intervention for Dachshund spine health. Excess weight increases the mechanical load on discs that are already compressed by the breed’s anatomy. A Dachshund at 12 pounds instead of 10 is not just 20% heavier; the force on each disc increases non-linearly with body weight. The National Research Council (NRC 2006) energy requirements for a 10-pound adult dog are roughly 400 kcal per day at maintenance. Many commercial Dachshund formulations are denser than that, and portion creep is common because Dachshunds are relentless scavengers. Measure portions carefully. Use a kitchen scale if you can. Treats should be no more than 10% of daily calories.

A large-breed puppy formula designed for slower growth is not appropriate for a Dachshund, which grows to adult size (standard up to 32 pounds; miniature 8 to 11 pounds, per AKC breed standard) within 8 to 10 months. A small-breed puppy formula with calcium between 1.0% and 1.8% and phosphorus between 0.8% and 1.6% on a dry-matter basis (NRC 2006 guidance) is the standard starting point. Avoid puppy formulas designed to maximize growth rate. A Dachshund that reaches adult size quickly and then maintains that size will have healthier discs than one that grows slowly then gains weight in middle age.

Once your Dachshund is an adult, a small-breed adult formula with moderate fat (12% to 15%) and high-quality protein (18% to 22%) supports lean-mass maintenance. Joint support ingredients like glucosamine and chondroitin sulfate are common in small-breed formulations but lack definitive evidence in dogs without active joint disease. If your Dachshund develops disc-bulge signs or clinical IVDD, work with your vet on a therapeutic diet designed for joint and spine support, often paired with restricted activity and medications like gabapentin or NSAIDs depending on severity.

Grain-free diets carry the same cardiac risk for Dachshunds as they do for other breeds. Adin et al. 2019 (JVIM 33:2691-2699) documented diet-associated dilated cardiomyopathy across multiple breeds fed certain grain-free formulations. A grain-inclusive, taurine-complete formulation is the safer default. Dachshunds are not a breed with particular reported susceptibility to DCM in the literature, but the general risk applies.

What we don’t know

The honest summary is that Dachshund genetics research has a severe data gap. The Donner 2023 cohort is the largest genetic survey of the breed, and it identified no Mendelian variants at observable carrier frequency. That finding is valuable, it means the breed is not burdened by the recessive-disease load that characterizes some breeds, but it also means we have almost no population-level data on disease incidence, age of onset, or penetrance for the conditions Dachshund owners encounter most.

IVDD is the defining health problem in Dachshunds. The breed carries the FGF4 retrogene (CFA12) at 18% allele frequency. This variant causes chondrodystrophy (CDDY) and is established as an IVDD risk factor across multiple breeds (Brown et al. 2017, PLOS Genetics). We do not yet know which Dachshunds with the variant will develop symptomatic disc disease and which will not. We do not have published prospective follow-up studies tracking cohorts of Dachshunds over their lifespans to map the interaction between the FGF4 variant, weight, activity level, age, and actual disease onset. The breed-club health programs focus on owner education about weight and activity, which is the right move, but the mechanistic research is still missing.

Cancer data, survivorship data, and cardiac screening prevalence are all absent from published peer-reviewed literature specific to Dachshunds. We know the breed’s median lifespan from atlas records is 13.6 years, which is longer than many larger breeds. Beyond that, the epidemiology is mostly unmapped.

Frequently asked questions about Dachshunds

Are Dachshunds prone to back problems? Yes. The breed’s long spine and short legs create mechanical load that predisposes to intervertebral disc disease. The FGF4 retrogene at 18% allele frequency is an additional genetic risk factor. Weight management and activity restriction (avoiding jumping from furniture) reduce risk substantially.

What is the most common health problem in Dachshunds? Intervertebral disc disease (IVDD), which ranges from mild disc bulging to severe extrusion causing hind-limb paralysis. Early signs include reluctance to jump, back pain, or dragging hind legs. Imaging and prompt veterinary care improve outcomes.

How long do Dachshunds live? The atlas-derived median lifespan is 13.6 years. Dachshunds are among the longest-lived dog breeds, and many live into their mid-to-late teens with good care.

Should I do a DNA test on my Dachshund? Dachshunds do not have a recommended Mendelian-disease panel because the breed has not shown observable carriers of single-gene recessive diseases at clinical frequency. Testing for the FGF4 retrogene is available but not yet routine, because penetrance for IVDD is incomplete and clinical guidelines for acting on a positive result are still developing.

What is the best diet for a Dachshund? A small-breed adult formula with moderate fat (12% to 15%), high-quality protein (18% to 22%), and grain inclusion is the standard. Weight management is more important than the specific formula. Keep portions measured, treats under 10% of daily calories, and maintain a lean body condition score to reduce spinal stress.

Are Dachshunds good with kids? Dachshunds are alert and affectionate but were originally bred as independent hunters. They can be territorial and may snap if startled or handled roughly. Supervision with young children is important. Teach children to approach calmly and not pick up the dog, which stresses the spine.

How much exercise does a Dachshund need? A healthy adult Dachshund needs 30 to 60 minutes of moderate exercise per day, usually satisfied by walks and indoor play. Avoid jumping from furniture or high-impact activities that stress the spine. Swimming is an excellent low-impact option.

Do Dachshunds shed a lot? Shedding varies by coat type. Smooth-coated Dachshunds shed year-round at moderate levels. Wirehaired and longhaired varieties shed less but require regular grooming to prevent matting.

A gift to human medicine

Dachshunds are a natural model for human disease

Because the same genes cause the same conditions across species, the inherited conditions documented in Dachshunds help researchers understand, and work toward treating, the human diseases they model. This is the dog advancing human medicine. The breed models the human disease; it does not have it, and this is not a prediction for your dog.

Human equivalents via OMIA → Mondo / OMIM. Model-of, not identity.
Documented in OMIA

Every condition recorded in the Dachshund

Beyond the testable carriers above, OMIA's literature catalogue records 24 genetic conditions in the Dachshund, 18 of which have a known human equivalent. This is the documented landscape across all Dachshunds ever studied, not a prediction for any one dog.

Plus 6 more conditions recorded in the Dachshund in OMIA.

Online Mendelian Inheritance in Animals (OMIA); Nicholas, Tammen & Sydney Informatics Hub, DOI 10.25910/2AMR-PV70
Documented in the breed's literature is not carrier status and not a forecast for an individual dog. Human equivalents are mapped via Mondo/OMIM. Carrier frequencies (above) are the separately-measured testable subset (Donner 2023).
The data behind this page

Where every number on this page came from.

This page draws on three primary data sources. Carrier frequencies for the Mendelian section come from Donner et al. 2023 (CC-BY-4.0). We grade these data at evidence Limited because the cohort is a direct-to-consumer ascertainment, which biases toward owners who chose to test their dogs. The panel also uses tag-SNP proxies for some variants rather than direct causal-variant assays. Limited is a study-design grade, not a quality grade: the Donner cohort is the largest open canine-genotype dataset in existence and we are grateful for it. We rate the confounding MEDIUM.

Population-genetic dimensions (heterozygosity, intra-breed PCA distance, nearest neighbors, trait-locus frequencies) come from CanVAS (Brundage 2026), harmonized through the Sniff Atlas. The exact release date and verification commit are pinned at the bottom of the page so a researcher can trace a number back to a specific snapshot. The disease-gene-variant graph comes from OMIA (Online Mendelian Inheritance in Animals; Nicholas, Tammen, and the Sydney Informatics Hub at the Sydney School of Veterinary Science, The University of Sydney; retrieved April 2026, DOI 10.25910/2AMR-PV70).

What this page does not yet have. Inheritance modes and per-disease penetrance evidence from Donner 2023 are now in the structured data for every variant the panel covers. Mondo, OMIM, Ensembl, and HGNC cross-references on gene pages remain pending, they arrive in December 2026 alongside the imputed 9.67M-variant CanVAS dataset via the OMIA SQL dump absorption. Until then, gene IDs carry NCBI Gene and OMIA phene URLs only; the wider human-homolog and disease-ontology cross-reference set fills in with that release.

How to cite this page. The computed dimensions on this page are derived from the open Sniff Atlas v1.0.1 (Gehring 2026, doi:10.5281/zenodo.20566358, CC-BY 4.0). Full citation formats including BibTeX, RIS, and CITATION.cff at sniff.world/cite.

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References
  1. Donner J, Freyer J, Davison S, Anderson H, Blades M, Honkanen L, et al. (2023). Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651. doi:10.1371/journal.pgen.1010651
  2. Brundage J, et al. (2026). CanVAS: a harmonized canine variant atlas. bioRxiv. doi:10.64898/2026.04.13.718238
  3. Nicholas, F.W., Tammen, I., & Sydney Informatics Hub. (2026). Online Mendelian Inheritance in Animals (OMIA) [dataset]. The University of Sydney. https://omia.org. doi:10.25910/2AMR-PV70 (retrieved April 2026).
Last updated
Sources: CanVAS (Brundage 2026) · Donner 2023 · OMIA