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Newfoundland

Newfoundland
Photo: Flickr user DanDee Shots / CC BY 2.0 · Wikimedia

182 Newfoundlands in the atlas. Every number on this page has a source.

Population-genetic snapshot of Newfoundlands in the Sniff Atlas, source-graded Mendelian carrier frequencies from Donner 2023, and nutrition guidance tied to the genetic findings above.

The plain version

Newfoundlands have a moderately diverse gene pool, which means there’s a good mix of genetic backgrounds within the breed. They are large dogs, typically weighing around 146 pounds, and usually live about 11 years. No specific health concerns showed up in genetic screenings, but it’s always a good idea to chat with your vet or consider testing for your own dog’s health.

What the atlas says about Newfoundland

In the atlas, the Newfoundland clusters consistently as Newfoundland (100% of the 182 dogs here). At the trait loci, SMOC2 runs lower than average (12% here vs 75%); ADAMTS17 runs higher than the atlas average (97% here vs 54%).

Genetic dimensions · CanVAS atlas

What the genome says about Newfoundland

Computed from the 18,477 research dogs in the Atlas.

Dogs in the Atlas
182Founders
74 from Hayward2016, 45 from Momozawa, 32 from Shannon
Genetic diversity
0.29Moderate
Mean heterozygosity across the breed. Ranks 38th most genetically tight of 107 ranked breeds.
What does genetic diversity mean?

How varied a breed's gene pool is — the share of gene spots where a typical dog of the breed carries two different versions rather than two identical ones.

How to read it: Higher = more diverse. Among well-sampled breeds it ranges roughly 0.22 (least diverse) to 0.33 (most diverse).

Diversity is a strength, not a verdict on any individual dog. Lower diversity means it's worth paying attention to recessive-risk testing — not that a dog is doomed.

Cluster structure
Splits into two genetic sub-populations
Intra-breed RMS distance: 28.40 · likely working/show-line, regional, or kennel lineage split.
What does within-breed variation mean?

How much individual dogs within the breed differ from each other genetically.

How to read it: Higher = more internal variety among individuals of the breed.

Sensitive to how many dogs of the breed we've sampled.

Related breeds
Gave rise to
In the Working group
Explore the full lineage map →
VBO foundation stock (breeding records) · AKC breed group
Relatedness is documented lineage + kennel family. Genetic-ancestry distance measures diversity, not kinship, so it isn't used here.
How long they live
11years (life expectancy)
95% CI 10.9–11.6 · VetCompass, McMillan 2024, n=1,529. source
What does typical lifespan mean?

The median age dogs of the breed tend to reach.

How to read it: Higher = longer-lived. Compare to longevity-for-size to see whether it's just a size effect.

Drawn from population lifespan records; individual dogs vary widely with care, genetics, and luck.

Trait genetics
Allele frequencies at named morphology loci

Frequency of the alternate allele in this breed at each locus's representative SNP.

Body size
IGF125%
HMGA282%
SMAD2100%
LCORL73%
STC251%
ADAMTS1797%
Leg length
FGF4·CFA1884%
FGF4·CFA1293%
Coat
RSPO262%
FGF536%
KRT7192%
MC1R46%
Ear set
MSRB395%
Skull shape
BMP391%
SMOC212%
n = 182 dogs · high confidence · CanVAS (Brundage 2026) · Sniff Atlas
Names & origins

Identified as Newfoundland (VBO:0200938) in the Vertebrate Breed Ontology (Mullen et al. 2025, CC-BY 4.0) · registry IDs FCI 50 · iDog 166 · VeNom 14476.

What you see when you look at a Newfoundland

What does the genome say about how a Newfoundland looks?

Newfoundlands look the way they do because of a small set of fixed and near-fixed morphology genes that, taken together, define the visible breed. Each translation below pairs the gene with the trait an owner actually sees, the breed's allele frequency at that locus, and a one-clause causal phrase.

Where the breed-defining genes act, mapped on a generic dog-body key — and how fixed each marker is in the Newfoundland. The figure is the most-settled marker we read in that region; the full per-locus panel is below. (The silhouette is a shared anatomical guide, not this breed's outline.)

Body sizeSMAD2 · 100%Skull shapeBMP3 · 91%EarsMSRB3 · 95%Leg lengthFGF4 CFA12 · 93%Coat & colorKRT71 · 92%
CanVAS trait-locus panel (Brundage 2026)
15 morphology markers read across 5 regions. Allele frequency = how fixed a marker is in this breed, not whether your dog carries it.

Size and build

IGF1 is at 25% for the small-body allele, leaving the breed firmly in the larger end of the dog body-size spectrum.

IGF1what this gene does

IGF1 is a gene that plays a key role in determining a dog's body size. It influences how much a dog grows, affecting overall stature.

For your dog: Knowing about IGF1 gives you insight into your dog's size traits, but it’s just one part of the bigger picture when it comes to their health and care.

Full IGF1 gene page →

HMGA2 sits at 82%. HMGA2 is a chromosome-10 size locus that acts together with IGF1, and intermediate frequencies reflect partial commitment to the dominant size variant.

HMGA2what this gene does

HMGA2 is a gene that influences body size in dogs, helping determine how big or small a dog grows.

For your dog: Knowing about HMGA2 helps you appreciate the genetic factors behind your dog's size, but it doesn't signal any health issues.

Full HMGA2 gene page →

SMAD2 is near-fixed at 100%, a chromosome-7 height locus differentiating small from giant breeds.

SMAD2what this gene does

SMAD2 is a gene involved in regulating body size by influencing how cells grow and develop.

For your dog: Knowing about SMAD2 helps understand your dog's size traits but isn't linked to health issues; no immediate action needed.

Full SMAD2 gene page →

LCORL sits at 73% at the NCAPG/LCORL height locus on chromosome 3.

LCORLwhat this gene does

LCORL is a gene that influences body size in dogs. It helps determine how big or small a dog might grow.

For your dog: Knowing about LCORL helps you appreciate the genetic factors behind your dog's size, but it’s just one piece of the bigger picture when it comes to health and care.

Full LCORL gene page →

STC2 sits at 51%.

ADAMTS17 is at 97%, near-fixed for the size variant.

ADAMTS17what this gene does

ADAMTS17 is a gene that influences body size and also plays a role in certain eye conditions. It affects the structure of tissues in the eye and elsewhere in the body.

For your dog: If your dog belongs to a breed known to carry ADAMTS17 variants, it’s worth discussing genetic testing and eye exams with your vet to stay ahead of potential issues.

Full ADAMTS17 gene page →

Leg length

The FGF4 retrogene on chromosome 18 sits at 84%. This is the leg-length variant. The intermediate frequency means some dogs in this breed carry the short-legged allele and some do not.

The FGF4 retrogene on chromosome 12 is near-fixed at 93%, the chondrodystrophic variant associated with intervertebral disc disease risk in breeds that carry it.

Coat type, length, and color

RSPO2 sits at 62% for the furnishings variant. Furnishings (the eyebrow-and-mustache pattern seen in Schnauzers and Wheaten Terriers) vary across the population at this intermediate frequency, and visible expression depends on the specific allele combination each dog carries.

RSPO2what this gene does

RSPO2 influences the texture and appearance of a dog's coat, particularly the presence of 'furnishings' like mustaches and eyebrows. It helps determine whether a dog has that distinctive wiry or textured look.

For your dog: If your dog has those wiry eyebrows or a mustache, RSPO2 is part of the reason—no health worries, just a coat feature worth knowing about.

Full RSPO2 gene page →

FGF5 sits at 36% for the long-coat variant. Coat length is influenced by other loci as well, so intermediate FGF5 frequencies do not always correspond to intermediate visible coat lengths.

FGF5what this gene does

FGF5 is a gene that influences the length of a dog's coat. It acts like a natural switch, telling hair follicles when to stop growing longer fur.

For your dog: If your dog has a notably long or short coat, FGF5 is likely part of the reason—no action needed, but it’s a neat genetic detail to know.

Full FGF5 gene page →

KRT71 is near-fixed at 92% for the wavy/curly variant. Coat curl phenotype varies across breeds at this fixation depending on modifier loci, and visible expression is not always curled even when the locus is fixed.

KRT71what this gene does

KRT71 is a gene that influences the curliness of a dog's coat. It helps determine whether a dog's fur is straight or has a distinctive curl.

For your dog: If your dog has a curly coat, KRT71 is likely part of the reason; it’s a natural variation, not a health concern.

Full KRT71 gene page →

MC1R sits at 46% at the representative SNP. MC1R controls the switch between red-to-gold pigment and black-to-brown pigment, with the e/e homozygous genotype producing the gold-to-red spectrum. Substrate frequencies at this SNP depend on the array's polarity, so visible coat color in the breed is a more reliable indicator than this single number.

MC1Rwhat this gene does

MC1R is a gene that influences coat color in dogs, affecting how pigments are produced in the fur.

For your dog: Knowing about MC1R gives insight into your dog's coat color but doesn't relate to health issues.

Full MC1R gene page →

Ears

MSRB3 is at 95% for the drop-ear allele, the genetic basis of the breed's signature dropped ear set.

MSRB3what this gene does

MSRB3 is a gene involved in the development of ear shape and structure in dogs.

For your dog: Understanding MSRB3 helps explain why your dog's ears look the way they do, but it isn't linked to any health issues.

Full MSRB3 gene page →

Skull shape

BMP3 is at 91%, contributing to the breed's brachycephalic skull shape.

BMP3what this gene does

BMP3 is a gene that influences the shape of a dog's skull, particularly contributing to a shorter, broader head shape known as brachycephaly.

For your dog: If your dog has a broad, short skull, it's worth discussing with your vet how this might impact their health, even though BMP3 isn't directly tied to illness.

Full BMP3 gene page →

SMOC2 is at 12%, leaving the breed in the long-headed dolichocephalic form.

SMOC2what this gene does

SMOC2 influences the shape of a dog's skull, particularly affecting how flat or short the face appears.

For your dog: If your dog has a short nose, it's worth discussing with your vet how this trait might impact their health over time.

Full SMOC2 gene page →
Mendelian-disease genetics

What genetic diseases do Newfoundlands carry?

From a panel of 250 Mendelian-disease variants screened in 1,054,293 dogs (Donner et al. 2023), Newfoundlands carry 4 of them at observable frequency. Carrier frequency is not clinical risk. Most recessive variants require two copies for disease expression; many dominant variants show incomplete penetrance. Read this as a population fingerprint of what's in the gene pool, not a per-dog prediction.

low 6.7%
n = 463 dogs · 3 variants tested · OMIA:000256-9615 · omia.org →
SLC3A1what this gene does

SLC3A1 is a gene that helps transport certain amino acids in the kidneys. It plays a key role in preventing the buildup of cystine, which can form stones.

For your dog: If your dog is from a breed known to carry SLC3A1 variants, it’s worth discussing cystinuria risks with your vet, especially if urinary issues arise.

Degenerative Myelopathy (DM)
Autosomal recessive (Incomplete penetrance)
low 1.1%
n = 463 dogs · 1 variant tested · OMIA:000263-9615 · omia.org →
SOD1what this gene does

SOD1 is a gene that helps protect cells from damage caused by harmful molecules called free radicals.

For your dog: If your dog is a carrier of SOD1 variants, it's worth discussing with your vet, but remember carrier status doesn't mean your dog will get the disease.

low 0.22%
n = 463 dogs · 1 variant tested · OMIA:001514-9615 · omia.org →
GDNFwhat this gene does

GDNF is a gene that helps support nerve cells, especially those involved in sensing pain and movement.

For your dog: If your dog is from a breed that can carry this gene change, it’s worth asking your vet about testing to understand any potential risks.

Cystinuria Type I-B (SLC7A9 p.A217T)
Autosomal recessive (Incomplete penetrance)
low 0.11%
n = 463 dogs · 2 variants tested · OMIA:001880-9615 · omia.org →
SLC7A9what this gene does

SLC7A9 is a gene that helps transport certain amino acids in the kidneys. It plays a role in how the body handles cystine, an amino acid that can form crystals.

For your dog: If your dog is a carrier, it’s worth discussing with your vet to monitor urinary health and catch any issues early.

Source: Donner J et al. 2023. Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651 · Evidence: Limited (DTC ascertainment, tag-SNP proxy) · Confounding MEDIUM · License CC-BY-4.0 · Phene IDs from OMIA (Sydney School of Veterinary Science, The University of Sydney; DOI 10.25910/2AMR-PV70).
Sample size in this breed: 463 dogs from the Donner 2023 cohort.

Which Mendelian variants matter most for Newfoundlands?

The Mendelian-disease table above lists 194 variants screened in 463 Newfoundlands, of which 4 were found at observable carrier frequency (Donner 2023). The carrier frequencies are low across the board. The breed sits at rank 38 of 107 for genetic diversity (lower rank indicates a tighter bottleneck), which is a separate measure from disease-variant burden but provides useful context for interpreting these frequencies. Two variants warrant attention.

Cystinuria Type I-A (SLC3A1)

Cystinuria Type I-A in Newfoundlands is an autosomal-recessive condition caused by a variant in SLC3A1. Affected dogs have excess urinary cystine excretion and are at risk for bladder-stone formation. The condition is manageable with diet and monitoring, though urinary obstructions from stones can become a medical emergency, particularly in males. About 6.7% of Newfoundlands in the Donner cohort carry one copy of the variant (n=463).

Testing is available through most commercial DNA labs and breed-specific panels. Breed-club health guidelines generally recommend screening breeding stock; check the Newfoundland Club of America’s official health pages (newfdogclub.org) for the most current protocol.

Degenerative Myelopathy (DM)

Degenerative Myelopathy in Newfoundlands is an autosomal-recessive condition with incomplete penetrance caused by a variant in SOD1. Affected dogs develop progressive hind-limb weakness and eventual paralysis, typically emerging in middle to late life. Only 1.1% of Newfoundlands in the Donner cohort carry the variant (n=463), making it rare in the breed.

The incomplete penetrance means not every dog with two copies becomes symptomatic. Testing is available and useful for breeding decisions in the small subset of carrier lines.

How should I test my Newfoundland?

A breed-specific panel covering at least SLC3A1 (cystinuria Type I-A) and SOD1 (degenerative myelopathy) is a reasonable baseline. Given the breed’s low overall variant burden, testing is most valuable for breeders rather than a routine wellness recommendation for pet owners.

What should I feed a Newfoundland?

Newfoundlands go from 2 pounds at birth to 130-150 pounds at adulthood in a compressed window of 18 to 24 months. The growth rate is so fast that the calcium-to-phosphorus ratio in the puppy formula is the single most important food decision an owner makes.

Large-breed puppy formulation is non-negotiable. The NRC 2006 nutrient requirements specify that growing giant-breed dogs need calcium at 1.2 to 1.8% of dry matter and a calcium-to-phosphorus ratio between 1.1:1 and 2:1 (NRC 2006). Oversupplementation with calcium in oversized portions is a reliable way to produce hip dysplasia, elbow dysplasia, and osteochondrosis in this breed. A puppy formula from a manufacturer that runs feeding trials and achieves those bounds is the starting point.

Portion control during the growth window is as important as the formula itself. Many Newfoundland owners underfeed their puppies because a 6-month-old already weighs 80 pounds and looks enormous. The vet’s growth chart is the actual guide, not the puppy’s size relative to adult dogs. Growth-stage caloric needs are higher than many owners expect, but structured correctly, not excessive. A veterinary growth chart, not a puppy’s size relative to adult dogs, is the right guide for adjusting portions at each stage.

Adult Newfoundlands benefit from lower-calorie, higher-fiber formulations relative to other giant breeds. The breed’s atlas-derived median lifespan is 11.0 years, which is moderate for the size category. Adult weight management becomes the dominant nutritional task after the growth window closes. Newfoundlands were bred for water work and stamina; a pet Newfoundland eating a maintenance kibble in a suburban yard is being fed for a job they aren’t doing. A lower-calorie, higher-fiber adult formula helps maintain lean body weight, which research consistently links to better joint health and longer life in large breeds.

The breed’s low cystinuria carrier frequency (6.7% for Type I-A, 0.11% for Type I-B; Donner 2023, n=463) does not warrant routine dietary intervention for most pets. Protein restriction is only indicated if a dog is a confirmed carrier with clinical evidence of stone formation. For the general population, a standard adult protein level (18-25% crude protein) is appropriate.

What we don’t know

The Newfoundland atlas contains 182 dogs, which is a moderate sample for a breed-specific health analysis. The Mendelian-variant burden is genuinely low, but that low frequency also means the confidence intervals around carrier percentages are wider than in larger-cohort breeds. A 6.7% estimate for cystinuria carries meaningful uncertainty in a cohort of 463; a breed-specific study with 2,000+ Newfoundlands would refine that number but does not yet exist.

Hip dysplasia is clinically common in Newfoundlands, but we lack a published, breed-specific epidemiological survey with OFA evaluation counts. The breed-club estimates differ on prevalence. A rigorous prevalence study would settle what owners are actually encountering.

Cancer appears in anecdotal breed-club discussions as a concern, but no published breed-specific cancer epidemiology exists for Newfoundlands. The honest summary is we do not have data on what cancers affect this breed, at what age they emerge, or what the breed’s cancer mortality rate is relative to other giant breeds.

Frequently asked questions about Newfoundlands

How long do Newfoundlands live? The atlas-derived median lifespan for Newfoundlands is 11.0 years, typical for a giant breed. Individual dogs vary widely; the atlas-derived figure reflects the median, not a ceiling or a floor.

What is the most common genetic disease in Newfoundlands? Cystinuria Type I-A is the most frequent Mendelian variant, present as a carrier in 6.7% of the breed (Donner 2023, n=463). Affected dogs can form bladder stones, but the condition is manageable with diet and monitoring.

Are Newfoundlands prone to hip dysplasia? Hip dysplasia is clinically common in the breed, but no breed-specific OFA prevalence study is published. Controlled growth during the puppy phase (proper calcium ratios, portion control, gradual exercise) is the strongest preventive measure.

Should I do a DNA test on my Newfoundland? For breeding stock, a panel covering cystinuria (SLC3A1 and SLC7A9) and degenerative myelopathy (SOD1) is worthwhile. For pet owners, testing is optional unless a dog has symptoms suggestive of cystinuria (recurrent urinary issues, bladder stones).

What should I feed my Newfoundland puppy? A large-breed puppy formula with calcium at 1.2 to 1.8% of dry matter and a calcium-to-phosphorus ratio between 1.1:1 and 2:1 is essential (NRC 2006). Stick to the manufacturer’s growth-stage portion recommendations even if your puppy looks large; controlled growth prevents skeletal disease.

What is the best diet for an adult Newfoundland? A lower-calorie, higher-fiber adult formula helps maintain lean weight, which research consistently links to better joint health and longer life in giant breeds. Newfoundlands were bred for water work and stamina; a pet eating a maintenance kibble needs fewer calories than the package assumes.

Are Newfoundlands good swimmers and water dogs? Yes. Newfoundlands were bred for water rescue and remain strong swimmers with water-resistant coats. Regular swimming is excellent low-impact exercise for joint health, though it does not replace controlled-pace land exercise during the growth window.

Do Newfoundlands have special coat care needs? Newfoundlands shed heavily year-round and blow their coat seasonally. Weekly brushing during heavy-shed periods prevents matting and reduces household hair. The coat itself does not require specialized feeding beyond the general calcium and growth management outlined above.

A gift to human medicine

Newfoundlands are a natural model for human disease

Because the same genes cause the same conditions across species, the inherited conditions documented in Newfoundlands help researchers understand, and work toward treating, the human diseases they model. This is the dog advancing human medicine. The breed models the human disease; it does not have it, and this is not a prediction for your dog.

Human equivalents via OMIA → Mondo / OMIM. Model-of, not identity.
Documented in OMIA

Every condition recorded in the Newfoundland

Beyond the testable carriers above, OMIA's literature catalogue records 10 genetic conditions in the Newfoundland, 7 of which have a known human equivalent. This is the documented landscape across all Newfoundlands ever studied, not a prediction for any one dog.

Online Mendelian Inheritance in Animals (OMIA); Nicholas, Tammen & Sydney Informatics Hub, DOI 10.25910/2AMR-PV70
Documented in the breed's literature is not carrier status and not a forecast for an individual dog. Human equivalents are mapped via Mondo/OMIM. Carrier frequencies (above) are the separately-measured testable subset (Donner 2023).
The data behind this page

Where every number on this page came from.

This page draws on three primary data sources. Carrier frequencies for the Mendelian section come from Donner et al. 2023 (CC-BY-4.0). We grade these data at evidence Limited because the cohort is a direct-to-consumer ascertainment, which biases toward owners who chose to test their dogs. The panel also uses tag-SNP proxies for some variants rather than direct causal-variant assays. Limited is a study-design grade, not a quality grade: the Donner cohort is the largest open canine-genotype dataset in existence and we are grateful for it. We rate the confounding MEDIUM.

Population-genetic dimensions (heterozygosity, intra-breed PCA distance, nearest neighbors, trait-locus frequencies) come from CanVAS (Brundage 2026), harmonized through the Sniff Atlas. The exact release date and verification commit are pinned at the bottom of the page so a researcher can trace a number back to a specific snapshot. The disease-gene-variant graph comes from OMIA (Online Mendelian Inheritance in Animals; Nicholas, Tammen, and the Sydney Informatics Hub at the Sydney School of Veterinary Science, The University of Sydney; retrieved April 2026, DOI 10.25910/2AMR-PV70).

What this page does not yet have. Inheritance modes and per-disease penetrance evidence from Donner 2023 are now in the structured data for every variant the panel covers. Mondo, OMIM, Ensembl, and HGNC cross-references on gene pages remain pending, they arrive in December 2026 alongside the imputed 9.67M-variant CanVAS dataset via the OMIA SQL dump absorption. Until then, gene IDs carry NCBI Gene and OMIA phene URLs only; the wider human-homolog and disease-ontology cross-reference set fills in with that release.

How to cite this page. The computed dimensions on this page are derived from the open Sniff Atlas v1.0.1 (Gehring 2026, doi:10.5281/zenodo.20566358, CC-BY 4.0). Full citation formats including BibTeX, RIS, and CITATION.cff at sniff.world/cite.

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References
  1. Donner J, Freyer J, Davison S, Anderson H, Blades M, Honkanen L, et al. (2023). Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651. doi:10.1371/journal.pgen.1010651
  2. Brundage J, et al. (2026). CanVAS: a harmonized canine variant atlas. bioRxiv. doi:10.64898/2026.04.13.718238
  3. Nicholas, F.W., Tammen, I., & Sydney Informatics Hub. (2026). Online Mendelian Inheritance in Animals (OMIA) [dataset]. The University of Sydney. https://omia.org. doi:10.25910/2AMR-PV70 (retrieved April 2026).
Last updated
Sources: CanVAS (Brundage 2026) · Donner 2023 · OMIA