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Shetland Sheepdog

Shetland Sheepdog
Photo: Flickr user nickobec (Nick Cowie) / CC BY-SA 2.0 · Wikimedia

37 Shetland Sheepdogs in the atlas. Every number on this page has a source.

Population-genetic snapshot of Shetland Sheepdogs in the Sniff Atlas, source-graded Mendelian carrier frequencies from Donner 2023, and nutrition guidance tied to the genetic findings above.

Also known as Dwarf Scotch Shepherd (obsolete), Sheltie, Shetland Collie (obsolete), and Toonie dog (obsolete).

The plain version

Shetland Sheepdogs come from a fairly tight gene pool, meaning they have some family similarities but still hold a good mix of traits. They’re small dogs, usually weighing around 19 lb, and often live about 14 years. Some Shelties carry genes related to medication sensitivity and eye issues, so it’s a good idea to talk with your vet about testing to keep your dog healthy.

What the atlas says about Shetland Sheepdog

In the atlas, the Shetland Sheepdog clusters consistently as Shetland Sheepdog (100% of the 37 dogs here). At the trait loci, HMGA2 runs lower than average (0% here vs 56%); ADAMTS17 runs higher than the atlas average (96% here vs 54%). Dogs here sit in a relatively sparse region of the atlas, fewer close neighbors than typical.

Ranks 23 of 107 on the bottleneck severity scale, well into the upper quartile of population contraction. High breed predictability score (2.29), individual dogs of this breed reliably cluster together genetically.

Median lifespan is 13.85 years, slightly longer than expected for the breed size (8.5 kg).

Genetic dimensions · CanVAS atlas

What the genome says about Shetland Sheepdog

Computed from the 18,477 research dogs in the Atlas.

Dogs in the Atlas
37Founders
25 from Hayward2016, 10 from Spatola, 1 from Shannon
Genetic diversity
0.27Tight
Mean heterozygosity across the breed. Ranks 23rd most genetically tight of 107 ranked breeds.
What does genetic diversity mean?

How varied a breed's gene pool is — the share of gene spots where a typical dog of the breed carries two different versions rather than two identical ones.

How to read it: Higher = more diverse. Among well-sampled breeds it ranges roughly 0.22 (least diverse) to 0.33 (most diverse).

Diversity is a strength, not a verdict on any individual dog. Lower diversity means it's worth paying attention to recessive-risk testing — not that a dog is doomed.

Cluster structure
Splits into two genetic sub-populations
Intra-breed RMS distance: 23.87 · likely working/show-line, regional, or kennel lineage split.
What does within-breed variation mean?

How much individual dogs within the breed differ from each other genetically.

How to read it: Higher = more internal variety among individuals of the breed.

Sensitive to how many dogs of the breed we've sampled.

Related breeds
In the Herding group
Explore the full lineage map →
VBO foundation stock (breeding records) · AKC breed group
Relatedness is documented lineage + kennel family. Genetic-ancestry distance measures diversity, not kinship, so it isn't used here.
How long they live
13.4years (life expectancy)
95% CI 13.1–13.6 · VetCompass, McMillan 2024, n=1,905. source
What does typical lifespan mean?

The median age dogs of the breed tend to reach.

How to read it: Higher = longer-lived. Compare to longevity-for-size to see whether it's just a size effect.

Drawn from population lifespan records; individual dogs vary widely with care, genetics, and luck.

Trait genetics
Allele frequencies at named morphology loci

Frequency of the alternate allele in this breed at each locus's representative SNP.

Body size
IGF171%
HMGA20%
SMAD2100%
LCORL100%
STC293%
ADAMTS1796%
Leg length
FGF4·CFA1887%
FGF4·CFA1285%
Coat
RSPO245%
FGF587%
KRT71100%
MC1R100%
Ear set
MSRB398%
Skull shape
BMP378%
SMOC269%
n = 37 dogs · moderate confidence · CanVAS (Brundage 2026) · Sniff Atlas
Names & origins

Other names

The Shetland Sheepdog is also recorded as Dwarf Scotch Shepherd (obsolete), Sheltie, Shetland Collie (obsolete), and Toonie dog (obsolete).

Identified as Shetland Sheepdog (VBO:0201217) in the Vertebrate Breed Ontology (Mullen et al. 2025, CC-BY 4.0) · registry IDs FCI 88 · iDog 216 · VeNom 14729.

Temperament

What Shetland Sheepdogs tend toward

Tendencies from owner surveys of purebred Shetland Sheepdogs — a leaning across the breed, not a prediction for any one dog. A bar’s strength shows how much of that behavior breed actually explains: for most it’s faint, because the rest is your dog, their training, and the life you give them.

Proximity Seekingbreed ~13%
affectionatealoof
Agonistic Thresholdbreed ~9%
assertivediffident
Biddabilitybreed ~18%
biddableindependent
Dog Sociabilitybreed ~8%
less sociablehighly sociable
Human Sociabilitybreed ~11%
less sociablehighly sociable
Environmental Engagementbreed ~9%
high engagementlow engagement
Arousal Levelbreed ~8%
arousedcomposed
Toy-directed Motor Patternsbreed ~18%
toy-directednot toy-directed
n = 39 dogs · Morrill et al. 2022, Science, Darwin's Ark (CC0)
Owner-reported purebreds; each factor n ≥ 25. "Breed ~%" is the share of this behavior explained by breed.
What you see when you look at a Shetland Sheepdog

What does the genome say about how a Shetland Sheepdog looks?

Shetland Sheepdogs look the way they do because of a small set of fixed and near-fixed morphology genes that, taken together, define the visible breed. Each translation below pairs the gene with the trait an owner actually sees, the breed's allele frequency at that locus, and a one-clause causal phrase.

Where the breed-defining genes act, mapped on a generic dog-body key — and how fixed each marker is in the Shetland Sheepdog. The figure is the most-settled marker we read in that region; the full per-locus panel is below. (The silhouette is a shared anatomical guide, not this breed's outline.)

Body sizeSMAD2 · 100%Skull shapeBMP3 · 78%EarsMSRB3 · 98%Leg lengthFGF4 CFA18 · 87%Coat & colorKRT71 · 100%
CanVAS trait-locus panel (Brundage 2026)
15 morphology markers read across 5 regions. Allele frequency = how fixed a marker is in this breed, not whether your dog carries it.

Size and build

IGF1 sits at 71% for the small-body allele. IGF1 is the gene that sets dog body size from Chihuahua to Great Dane. Intermediate frequencies typically keep a breed in the mid-sized range rather than tipping toward the larger working forms.

IGF1what this gene does

IGF1 is a gene that plays a key role in determining a dog's body size. It influences how much a dog grows, affecting overall stature.

For your dog: Knowing about IGF1 gives you insight into your dog's size traits, but it’s just one part of the bigger picture when it comes to their health and care.

Full IGF1 gene page →

HMGA2 is at 0%, leaving most of the size signal to other loci in the panel.

HMGA2what this gene does

HMGA2 is a gene that influences body size in dogs, helping determine how big or small a dog grows.

For your dog: Knowing about HMGA2 helps you appreciate the genetic factors behind your dog's size, but it doesn't signal any health issues.

Full HMGA2 gene page →

SMAD2 is near-fixed at 100%, a chromosome-7 height locus differentiating small from giant breeds.

SMAD2what this gene does

SMAD2 is a gene involved in regulating body size by influencing how cells grow and develop.

For your dog: Knowing about SMAD2 helps understand your dog's size traits but isn't linked to health issues; no immediate action needed.

Full SMAD2 gene page →

LCORL is near-fixed at 100%, the NCAPG/LCORL height locus that is one of the strongest single contributors to canine body size.

LCORLwhat this gene does

LCORL is a gene that influences body size in dogs. It helps determine how big or small a dog might grow.

For your dog: Knowing about LCORL helps you appreciate the genetic factors behind your dog's size, but it’s just one piece of the bigger picture when it comes to health and care.

Full LCORL gene page →

STC2 is near-fixed at 93%, modulating growth-axis signaling toward the breed's body-size set point.

ADAMTS17 is at 96%, near-fixed for the size variant.

ADAMTS17what this gene does

ADAMTS17 is a gene that influences body size and also plays a role in certain eye conditions. It affects the structure of tissues in the eye and elsewhere in the body.

For your dog: If your dog belongs to a breed known to carry ADAMTS17 variants, it’s worth discussing genetic testing and eye exams with your vet to stay ahead of potential issues.

Full ADAMTS17 gene page →

Leg length

The FGF4 retrogene on chromosome 18 is near-fixed in this breed at 87%. This is the leg-length variant. The breed is fully committed to the long-legged form rather than the short-legged Corgi-and-Dachshund body plan.

The FGF4 retrogene on chromosome 12 is near-fixed at 85%, the chondrodystrophic variant associated with intervertebral disc disease risk in breeds that carry it.

Coat type, length, and color

RSPO2 sits at 45% for the furnishings variant. Furnishings (the eyebrow-and-mustache pattern seen in Schnauzers and Wheaten Terriers) vary across the population at this intermediate frequency, and visible expression depends on the specific allele combination each dog carries.

RSPO2what this gene does

RSPO2 influences the texture and appearance of a dog's coat, particularly the presence of 'furnishings' like mustaches and eyebrows. It helps determine whether a dog has that distinctive wiry or textured look.

For your dog: If your dog has those wiry eyebrows or a mustache, RSPO2 is part of the reason—no health worries, just a coat feature worth knowing about.

Full RSPO2 gene page →

FGF5 is at 87% for the long-coat variant, which is why the breed's coat sits where it does on the long end of the dog coat-length spectrum.

FGF5what this gene does

FGF5 is a gene that influences the length of a dog's coat. It acts like a natural switch, telling hair follicles when to stop growing longer fur.

For your dog: If your dog has a notably long or short coat, FGF5 is likely part of the reason—no action needed, but it’s a neat genetic detail to know.

Full FGF5 gene page →

KRT71 is near-fixed at 100% for the wavy/curly variant. Coat curl phenotype varies across breeds at this fixation depending on modifier loci, and visible expression is not always curled even when the locus is fixed.

KRT71what this gene does

KRT71 is a gene that influences the curliness of a dog's coat. It helps determine whether a dog's fur is straight or has a distinctive curl.

For your dog: If your dog has a curly coat, KRT71 is likely part of the reason; it’s a natural variation, not a health concern.

Full KRT71 gene page →

MC1R is at 100% at the representative SNP. MC1R controls the switch between red-to-gold and black-to-brown pigment, with the e/e homozygous genotype producing the gold-to-red spectrum by blocking eumelanin (black and brown pigment).

MC1Rwhat this gene does

MC1R is a gene that influences coat color in dogs, affecting how pigments are produced in the fur.

For your dog: Knowing about MC1R gives insight into your dog's coat color but doesn't relate to health issues.

Full MC1R gene page →

Ears

MSRB3 is at 98% for the drop-ear allele, the genetic basis of the breed's signature dropped ear set.

MSRB3what this gene does

MSRB3 is a gene involved in the development of ear shape and structure in dogs.

For your dog: Understanding MSRB3 helps explain why your dog's ears look the way they do, but it isn't linked to any health issues.

Full MSRB3 gene page →

Skull shape

BMP3 sits at 78%, contributing to the breed's moderate, mesaticephalic head shape rather than the extreme brachycephalic form.

BMP3what this gene does

BMP3 is a gene that influences the shape of a dog's skull, particularly contributing to a shorter, broader head shape known as brachycephaly.

For your dog: If your dog has a broad, short skull, it's worth discussing with your vet how this might impact their health, even though BMP3 isn't directly tied to illness.

Full BMP3 gene page →

SMOC2 sits at 69%, contributing to the breed's moderate head shape.

SMOC2what this gene does

SMOC2 influences the shape of a dog's skull, particularly affecting how flat or short the face appears.

For your dog: If your dog has a short nose, it's worth discussing with your vet how this trait might impact their health over time.

Full SMOC2 gene page →
Mendelian-disease genetics

What genetic diseases do Shetland Sheepdogs carry?

From a panel of 250 Mendelian-disease variants screened in 1,054,293 dogs (Donner et al. 2023), Shetland Sheepdogs carry 7 of them at observable frequency. Carrier frequency is not clinical risk. Most recessive variants require two copies for disease expression; many dominant variants show incomplete penetrance. Read this as a population fingerprint of what's in the gene pool, not a per-dog prediction.

n = 945 dogs · 1 variant tested · OMIA:001402-9615 · omia.org →
ABCB1what this gene does

ABCB1 is a gene that helps control how certain drugs are processed and cleared from a dog's body.

For your dog: If your dog is from a breed that carries this gene variant, ask your vet about medication sensitivities before giving any new drugs.

Collie Eye Anomaly (CEA)
Autosomal recessive
moderate 12.5%
n = 945 dogs · 1 variant tested · OMIA:000218-9615 · omia.org →
NHEJ1what this gene does

NHEJ1 is a gene involved in repairing breaks in DNA, helping maintain the integrity of genetic information in cells.

For your dog: If your dog belongs to one of the breeds known to carry this gene variant, it's worth discussing testing with your vet to understand any potential eye health risks.

Degenerative Myelopathy (DM)
Autosomal recessive (Incomplete penetrance)
low 9.1%
n = 945 dogs · 1 variant tested · OMIA:000263-9615 · omia.org →
SOD1what this gene does

SOD1 is a gene that helps protect cells from damage caused by harmful molecules called free radicals.

For your dog: If your dog is a carrier of SOD1 variants, it's worth discussing with your vet, but remember carrier status doesn't mean your dog will get the disease.

n = 944 dogs · 1 variant tested · OMIA:001298-9615 · omia.org →
PRCDwhat this gene does

PRCD is a gene involved in the health of a dog's retina, the part of the eye that detects light and helps with vision.

For your dog: If your dog belongs to a breed known to carry PRCD changes, it's worth discussing eye health and potential genetic testing with your vet.

Source: Donner J et al. 2023. Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651 · Evidence: Limited (DTC ascertainment, tag-SNP proxy) · Confounding MEDIUM · License CC-BY-4.0 · Phene IDs from OMIA (Sydney School of Veterinary Science, The University of Sydney; DOI 10.25910/2AMR-PV70).
Sample size in this breed: 945 dogs from the Donner 2023 cohort.

Which Mendelian variants matter most for Shetland Sheepdogs?

The Mendelian-disease table above lists 7 variants at observable carrier frequency, from 194 variants screened across 945 Shetland Sheepdogs (Donner 2023). Two account for the breed’s highest carrier frequencies and the most pressing testing decisions.

Medication Sensitivity (MDR1)

Medication Sensitivity in Shetland Sheepdogs is an autosomal-dominant condition caused by a loss-of-function variant in the ABCB1 gene (commonly called MDR1). The variant impairs the blood-brain barrier’s ability to pump certain drugs out of the central nervous system, making affected dogs hypersensitive to common antiparasitics (ivermectin, milbemycin) and a handful of other medications. 11.1% of Shetland Sheepdogs in the Donner cohort carry the variant (n=945).

In the Donner 2023 phenotype-confirmed dataset, 1 of 2 at-risk dogs showed clinical signs, suggesting penetrance is not fully predictable from genotype alone (Donner 2023). Not every carrier will be clinically affected. Testing is widely available. The practical impact is straightforward: if your Shetland carries MDR1, your vet can choose alternative antiparasitics. The Collie Health Foundation maintains a detailed MDR1 drug list.

Collie Eye Anomaly (CEA)

Collie Eye Anomaly in Shetland Sheepdogs is an autosomal-recessive developmental eye condition. The retinal layering is abnormal and the choroid may be hypoplastic or absent in patches. Most affected dogs show no clinical signs and retain normal vision for life. Some experience more severe intraocular hemorrhage or detachment. 12.5% of Shetland Sheepdogs in the Donner cohort carry the variant (n=945).

CEA is breed-specific in its expression and prevalence. Testing is available through the Canine Eye Registration Foundation (CERF) as part of a clinical eye exam, and DNA testing is available through several labs. The Shetland Sheepdog breed-club health recommendations stress pre-breeding screening.

Degenerative Myelopathy (DM)

Degenerative Myelopathy in Shetland Sheepdogs is an autosomal-recessive, late-onset neurodegenerative condition with incomplete penetrance. Affected dogs develop progressive hind-limb weakness beginning in middle age and may eventually lose mobility. 9.1% of Shetland Sheepdogs carry the variant (n=945).

Only a fraction of dogs with two copies develop clinical signs, which defines the incomplete penetrance. Testing is available. Dogs with a positive genetic status should have veterinary monitoring for early signs of incoordination or weakness.

How should I test my Shetland Sheepdog?

A breed-specific Mendelian panel is the appropriate first step. The minimum set covers MDR1 (medication sensitivity), CEA (Collie eye anomaly), and DM (degenerative myelopathy). Additional rare retinal conditions, BBS2-PRA, vWD 3, CNGA1-PRA, and prcd-PRA, are lower frequency but available through full-breed panels. Most commercial CLIA-accredited labs offer a Shetland Sheepdog or Collie-group panel that covers the top three.

What should I feed a Shetland Sheepdog?

Shetland Sheepdogs were built for sustained herding work in Scottish hill country, and that metabolic expectation still shapes what they need. A working Sheltie moving stock for six hours needs a different fuel strategy than a pet Sheltie in a suburban house, and the food choice has to match the dog’s actual job.

For working and high-activity Shelties, caloric density and protein concentration matter most. Shetland Sheepdogs are built for endurance, not bulk. A 15-pound herding dog burning calories for hours should eat a food in the 4.0 to 4.5 kcal/gram range with protein at 20% to 25% of calories, well-balanced in amino acids. Working-dog formulas from major manufacturers (Purina Pro Plan, Royal Canin, Orijen) calibrate to that window and report taurine content. NRC 2006 guidelines for dogs cite 18% crude protein as the minimum for maintenance; actively working Shelties benefit from the 22 to 25% range (NRC, Nutrient Requirements of Dogs and Cats, 2006).

For pet Shelties in moderate activity, maintenance kibble with careful portion control prevents weight gain. The breed’s size means portion drift is invisible, half a cup versus three-quarters of a cup seems trivial until your 15-pound dog becomes 18 pounds, which moves metabolic wear upward. Obesity is a leading contributor to early joint problems in small-breed dogs (NRC 2006). A maintenance formula (12 to 15% calories from protein) fed by weight-based volume at the manufacturer’s recommendation, adjusted downward if the dog gains visibly, is the stable path.

Coat quality in Shelties depends on omega-3 and omega-6 balance more than breed-specific genetics. The breed’s double coat reflects high rates of long-coat variants: 87% carry the FGF5 long-coat allele and 45% carry the RSPO2 furnishings variant in the atlas cohort. A food with fish-based omega-3 and a balanced omega-6 to omega-3 ratio (ideally between 5:1 and 10:1) keeps the undercoat dense and the guard coat lustrous. Most grain-inclusive adult formulas meet this range; grain-free formulas sometimes do not.

Avoid grain-free diets unless there is a documented food allergy. The FDA has not issued a breed-specific cardiac advisory for Shetland Sheepdogs as it has for Golden Retrievers and some other breeds, but the mechanistic concern around taurine bioavailability in pulse-heavy formulations applies to all breeds. If grain-free is medically necessary, choose formulas with reported taurine testing and discuss the choice with your vet. Stick with grain-inclusive formulas or, if grain-free is medically necessary, choose formulas with reported taurine testing and vet approval.

What we don’t know

The incomplete penetrance in Degenerative Myelopathy and MDR1 medication sensitivity leaves open the question of which additional genetic or environmental factors determine whether a carrier becomes clinically affected. A Shetland Sheepdog with the DM variant may never develop signs; another may start showing symptoms at age 8. We do not yet have predictive markers that separate those trajectories.

The breed’s atlas cohort is small (37 dogs), which means genetic and lifespan data are provisional. The median lifespan from the atlas is 13.8 years, and the breed-club estimate aligns with that window, but variation around that mean is large and individual longevity outliers are not yet well-characterized. The atlas cohort for Shelties will grow as more dogs are enrolled, and estimates will sharpen with larger samples.

The atlas also shows two sub-populations in the current Shetland Sheepdog sample, suggesting some stratification in the breed’s ancestry, but the practical implications of that structure for health or breeding decisions are not yet mapped.

Frequently asked questions about Shetland Sheepdogs

What is the most common genetic disease in Shetland Sheepdogs? Collie Eye Anomaly and medication sensitivity (MDR1) are tied for highest carrier frequency in the breed. CEA affects 12.5% of Shelties as carriers in the Donner cohort (Donner 2023, n=945). Most affected dogs have normal vision for life, though some develop more severe intraocular changes.

Are Shetland Sheepdogs sensitive to medications? About 11.1% carry a variant in MDR1 that makes them hypersensitive to certain antiparasitic drugs and a few other medications (Donner 2023, n=945). If your Sheltie tests positive, your vet can use alternative antiparasitics. The Collie Health Foundation maintains a current drug list.

How long do Shetland Sheepdogs live? The atlas median lifespan is 13.8 years, consistent with breed-club estimates. Individual Shelties live well into their late teens with good care, while others may not reach 13. Weight management and regular vet checkups are the best levers for longevity in this breed.

Should I do a DNA test on my Shetland Sheepdog? For breeding stock, yes. Test for MDR1, Collie Eye Anomaly, and Degenerative Myelopathy. Additional retinal variants are available on full-breed panels but are much rarer. Pet Shelties benefit from knowing MDR1 status to guide medication choices with your vet.

What is the best diet for a Shetland Sheepdog? It depends on activity level. A working Sheltie needs higher calories and protein (4.0 to 4.5 kcal/gram, 22 to 25% protein); a pet Sheltie does well on a standard maintenance formula with careful portion control. Both benefit from grain-inclusive, fish-oil-balanced kibble unless there is a documented allergy.

Are Shetland Sheepdogs good with kids? Shetland Sheepdogs are herding dogs and may nip at heels during play, especially young dogs that haven’t learned to inhibit that instinct. Socialization and management around small children matter. They are loyal and affectionate family members when raised with kids from puppyhood.

Do Shetland Sheepdogs require a lot of exercise? Yes. The breed was built for all-day herding and retains high drive in most lines. Shelties without adequate exercise, a minimum of one hour of active play or work daily, develop anxiety, destructiveness, and behavior problems. Mental stimulation (training, puzzle work) counts as much as physical activity.

What should I know about Shetland Sheepdog temperament? Shelties are intelligent, eager to please, and highly sensitive to handler emotion and tone. They are prone to sound sensitivity (vacuum cleaners, fireworks) and can develop anxiety if not socialized early. Training works best with positive reinforcement and consistency; harsh corrections often backfire in this breed.

A gift to human medicine

Shetland Sheepdogs are a natural model for human disease

Because the same genes cause the same conditions across species, the inherited conditions documented in Shetland Sheepdogs help researchers understand, and work toward treating, the human diseases they model. This is the dog advancing human medicine. The breed models the human disease; it does not have it, and this is not a prediction for your dog.

Human equivalents via OMIA → Mondo / OMIM. Model-of, not identity.
Documented in OMIA

Every condition recorded in the Shetland Sheepdog

Beyond the testable carriers above, OMIA's literature catalogue records 17 genetic conditions in the Shetland Sheepdog, 14 of which have a known human equivalent. This is the documented landscape across all Shetland Sheepdogs ever studied, not a prediction for any one dog.

Online Mendelian Inheritance in Animals (OMIA); Nicholas, Tammen & Sydney Informatics Hub, DOI 10.25910/2AMR-PV70
Documented in the breed's literature is not carrier status and not a forecast for an individual dog. Human equivalents are mapped via Mondo/OMIM. Carrier frequencies (above) are the separately-measured testable subset (Donner 2023).
The data behind this page

Where every number on this page came from.

This page draws on three primary data sources. Carrier frequencies for the Mendelian section come from Donner et al. 2023 (CC-BY-4.0). We grade these data at evidence Limited because the cohort is a direct-to-consumer ascertainment, which biases toward owners who chose to test their dogs. The panel also uses tag-SNP proxies for some variants rather than direct causal-variant assays. Limited is a study-design grade, not a quality grade: the Donner cohort is the largest open canine-genotype dataset in existence and we are grateful for it. We rate the confounding MEDIUM.

Population-genetic dimensions (heterozygosity, intra-breed PCA distance, nearest neighbors, trait-locus frequencies) come from CanVAS (Brundage 2026), harmonized through the Sniff Atlas. The exact release date and verification commit are pinned at the bottom of the page so a researcher can trace a number back to a specific snapshot. The disease-gene-variant graph comes from OMIA (Online Mendelian Inheritance in Animals; Nicholas, Tammen, and the Sydney Informatics Hub at the Sydney School of Veterinary Science, The University of Sydney; retrieved April 2026, DOI 10.25910/2AMR-PV70).

What this page does not yet have. Inheritance modes and per-disease penetrance evidence from Donner 2023 are now in the structured data for every variant the panel covers. Mondo, OMIM, Ensembl, and HGNC cross-references on gene pages remain pending, they arrive in December 2026 alongside the imputed 9.67M-variant CanVAS dataset via the OMIA SQL dump absorption. Until then, gene IDs carry NCBI Gene and OMIA phene URLs only; the wider human-homolog and disease-ontology cross-reference set fills in with that release.

How to cite this page. The computed dimensions on this page are derived from the open Sniff Atlas v1.0.1 (Gehring 2026, doi:10.5281/zenodo.20566358, CC-BY 4.0). Full citation formats including BibTeX, RIS, and CITATION.cff at sniff.world/cite.

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References
  1. Donner J, Freyer J, Davison S, Anderson H, Blades M, Honkanen L, et al. (2023). Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651. doi:10.1371/journal.pgen.1010651
  2. Brundage J, et al. (2026). CanVAS: a harmonized canine variant atlas. bioRxiv. doi:10.64898/2026.04.13.718238
  3. Nicholas, F.W., Tammen, I., & Sydney Informatics Hub. (2026). Online Mendelian Inheritance in Animals (OMIA) [dataset]. The University of Sydney. https://omia.org. doi:10.25910/2AMR-PV70 (retrieved April 2026).
Last updated
Sources: CanVAS (Brundage 2026) · Donner 2023 · OMIA