Skip to main content
snıff

Siberian Husky

Siberian Husky
Photo: xJaM (talk · contribs) / CC BY-SA 3.0 · Wikimedia

30 Siberian Huskys in the atlas. Every number on this page has a source.

Population-genetic snapshot of Siberian Huskys in the Sniff Atlas, source-graded Mendelian carrier frequencies from Donner 2023, and nutrition guidance tied to the genetic findings above.

Also known as Chukcha, Chuksha, Husky, and Sibe.

The plain version

Siberian Huskies have a moderately diverse gene pool, meaning they have a good mix of genetic background without being too inbred. They typically weigh around 47 pounds and live about 12 years on average. Huskies are known for their striking looks and friendly, energetic nature. No specific genetic health concerns were found in this breed’s gene pool based on current screening, but regular vet check-ups are always a great idea.

What the atlas says about Siberian Husky

In the atlas, the Siberian Husky clusters consistently as Siberian Husky (100% of the 30 dogs here). At the trait loci, ADAMTS17 runs lower than average (10% here vs 54%); MC1R runs higher than the atlas average (100% here vs 62%). Dogs here sit in a relatively sparse region of the atlas, fewer close neighbors than typical.

High breed predictability score (1.57), individual dogs of this breed reliably cluster together genetically.

Genetic dimensions · CanVAS atlas

What the genome says about Siberian Husky

Computed from the 18,477 research dogs in the Atlas.

Dogs in the Atlas
30Founders
17 from Hayward2016, 10 from Spatola, 3 from JenkinsWGS
Genetic diversity
0.31Moderate
Mean heterozygosity across the breed. Ranks 54th most genetically tight of 107 ranked breeds.
What does genetic diversity mean?

How varied a breed's gene pool is — the share of gene spots where a typical dog of the breed carries two different versions rather than two identical ones.

How to read it: Higher = more diverse. Among well-sampled breeds it ranges roughly 0.22 (least diverse) to 0.33 (most diverse).

Diversity is a strength, not a verdict on any individual dog. Lower diversity means it's worth paying attention to recessive-risk testing — not that a dog is doomed.

Cluster structure
Splits into two genetic sub-populations
Intra-breed RMS distance: 24.79 · likely working/show-line, regional, or kennel lineage split.
What does within-breed variation mean?

How much individual dogs within the breed differ from each other genetically.

How to read it: Higher = more internal variety among individuals of the breed.

Sensitive to how many dogs of the breed we've sampled.

Related breeds
In the Working group
Explore the full lineage map →
VBO foundation stock (breeding records) · AKC breed group
Relatedness is documented lineage + kennel family. Genetic-ancestry distance measures diversity, not kinship, so it isn't used here.
How long they live
11.9years (life expectancy)
95% CI 11.7–12 · VetCompass, McMillan 2024, n=4,453. source
What does typical lifespan mean?

The median age dogs of the breed tend to reach.

How to read it: Higher = longer-lived. Compare to longevity-for-size to see whether it's just a size effect.

Drawn from population lifespan records; individual dogs vary widely with care, genetics, and luck.

Trait genetics
Allele frequencies at named morphology loci

Frequency of the alternate allele in this breed at each locus's representative SNP.

Body size
IGF180%
HMGA292%
SMAD285%
LCORL93%
STC254%
ADAMTS1710%
Leg length
FGF4·CFA1878%
FGF4·CFA1283%
Coat
RSPO266%
FGF598%
KRT7198%
MC1R100%
Ear set
MSRB395%
Skull shape
BMP371%
SMOC288%
n = 30 dogs · moderate confidence · CanVAS (Brundage 2026) · Sniff Atlas
Names & origins

Other names

The Siberian Husky is also recorded as Chukcha, Chuksha, Husky, and Sibe.

Identified as Siberian Husky (VBO:0201233) in the Vertebrate Breed Ontology (Mullen et al. 2025, CC-BY 4.0) · registry IDs FCI 270 · iDog 220 · VeNom 14275.

What you see when you look at a Siberian Husky

What does the genome say about how a Siberian Husky looks?

Siberian Huskys look the way they do because of a small set of fixed and near-fixed morphology genes that, taken together, define the visible breed. Each translation below pairs the gene with the trait an owner actually sees, the breed's allele frequency at that locus, and a one-clause causal phrase.

Where the breed-defining genes act, mapped on a generic dog-body key — and how fixed each marker is in the Siberian Husky. The figure is the most-settled marker we read in that region; the full per-locus panel is below. (The silhouette is a shared anatomical guide, not this breed's outline.)

Body sizeLCORL · 93%Skull shapeSMOC2 · 88%EarsMSRB3 · 95%Leg lengthFGF4 CFA12 · 83%Coat & colorMC1R · 100%
CanVAS trait-locus panel (Brundage 2026)
15 morphology markers read across 5 regions. Allele frequency = how fixed a marker is in this breed, not whether your dog carries it.

Size and build

IGF1 sits at 80% for the small-body allele. IGF1 is the gene that sets dog body size from Chihuahua to Great Dane. Intermediate frequencies typically keep a breed in the mid-sized range rather than tipping toward the larger working forms.

IGF1what this gene does

IGF1 is a gene that plays a key role in determining a dog's body size. It influences how much a dog grows, affecting overall stature.

For your dog: Knowing about IGF1 gives you insight into your dog's size traits, but it’s just one part of the bigger picture when it comes to their health and care.

Full IGF1 gene page →

HMGA2 is near-fixed at 92%, reinforcing the breed's size signal through a second locus on chromosome 10.

HMGA2what this gene does

HMGA2 is a gene that influences body size in dogs, helping determine how big or small a dog grows.

For your dog: Knowing about HMGA2 helps you appreciate the genetic factors behind your dog's size, but it doesn't signal any health issues.

Full HMGA2 gene page →

SMAD2 is near-fixed at 85%, a chromosome-7 height locus differentiating small from giant breeds.

SMAD2what this gene does

SMAD2 is a gene involved in regulating body size by influencing how cells grow and develop.

For your dog: Knowing about SMAD2 helps understand your dog's size traits but isn't linked to health issues; no immediate action needed.

Full SMAD2 gene page →

LCORL is near-fixed at 93%, the NCAPG/LCORL height locus that is one of the strongest single contributors to canine body size.

LCORLwhat this gene does

LCORL is a gene that influences body size in dogs. It helps determine how big or small a dog might grow.

For your dog: Knowing about LCORL helps you appreciate the genetic factors behind your dog's size, but it’s just one piece of the bigger picture when it comes to health and care.

Full LCORL gene page →

STC2 sits at 54%.

ADAMTS17 is at 10%, the lower-frequency allele in this breed.

ADAMTS17what this gene does

ADAMTS17 is a gene that influences body size and also plays a role in certain eye conditions. It affects the structure of tissues in the eye and elsewhere in the body.

For your dog: If your dog belongs to a breed known to carry ADAMTS17 variants, it’s worth discussing genetic testing and eye exams with your vet to stay ahead of potential issues.

Full ADAMTS17 gene page →

Leg length

The FGF4 retrogene on chromosome 18 sits at 78%. This is the leg-length variant. The intermediate frequency means some dogs in this breed carry the short-legged allele and some do not.

The FGF4 retrogene on chromosome 12 sits at 83%, the chondrodystrophic variant.

Coat type, length, and color

RSPO2 sits at 66% for the furnishings variant. Furnishings (the eyebrow-and-mustache pattern seen in Schnauzers and Wheaten Terriers) vary across the population at this intermediate frequency, and visible expression depends on the specific allele combination each dog carries.

RSPO2what this gene does

RSPO2 influences the texture and appearance of a dog's coat, particularly the presence of 'furnishings' like mustaches and eyebrows. It helps determine whether a dog has that distinctive wiry or textured look.

For your dog: If your dog has those wiry eyebrows or a mustache, RSPO2 is part of the reason—no health worries, just a coat feature worth knowing about.

Full RSPO2 gene page →

FGF5 is at 98% for the long-coat variant, which is why the breed's coat sits where it does on the long end of the dog coat-length spectrum.

FGF5what this gene does

FGF5 is a gene that influences the length of a dog's coat. It acts like a natural switch, telling hair follicles when to stop growing longer fur.

For your dog: If your dog has a notably long or short coat, FGF5 is likely part of the reason—no action needed, but it’s a neat genetic detail to know.

Full FGF5 gene page →

KRT71 is near-fixed at 98% for the wavy/curly variant. Coat curl phenotype varies across breeds at this fixation depending on modifier loci, and visible expression is not always curled even when the locus is fixed.

KRT71what this gene does

KRT71 is a gene that influences the curliness of a dog's coat. It helps determine whether a dog's fur is straight or has a distinctive curl.

For your dog: If your dog has a curly coat, KRT71 is likely part of the reason; it’s a natural variation, not a health concern.

Full KRT71 gene page →

MC1R is at 100% at the representative SNP. MC1R controls the switch between red-to-gold and black-to-brown pigment, with the e/e homozygous genotype producing the gold-to-red spectrum by blocking eumelanin (black and brown pigment).

MC1Rwhat this gene does

MC1R is a gene that influences coat color in dogs, affecting how pigments are produced in the fur.

For your dog: Knowing about MC1R gives insight into your dog's coat color but doesn't relate to health issues.

Full MC1R gene page →

Ears

MSRB3 is at 95% for the drop-ear allele, the genetic basis of the breed's signature dropped ear set.

MSRB3what this gene does

MSRB3 is a gene involved in the development of ear shape and structure in dogs.

For your dog: Understanding MSRB3 helps explain why your dog's ears look the way they do, but it isn't linked to any health issues.

Full MSRB3 gene page →

Skull shape

BMP3 sits at 71%, contributing to the breed's moderate, mesaticephalic head shape rather than the extreme brachycephalic form.

BMP3what this gene does

BMP3 is a gene that influences the shape of a dog's skull, particularly contributing to a shorter, broader head shape known as brachycephaly.

For your dog: If your dog has a broad, short skull, it's worth discussing with your vet how this might impact their health, even though BMP3 isn't directly tied to illness.

Full BMP3 gene page →

SMOC2 is at 88%, the major locus contributing to the breed's brachycephalic face shape.

SMOC2what this gene does

SMOC2 influences the shape of a dog's skull, particularly affecting how flat or short the face appears.

For your dog: If your dog has a short nose, it's worth discussing with your vet how this trait might impact their health over time.

Full SMOC2 gene page →
Mendelian-disease genetics

What genetic diseases do Siberian Huskys carry?

From a panel of 250 Mendelian-disease variants screened in 1,054,293 dogs (Donner et al. 2023), Siberian Huskys carry 28 of them at observable frequency. Carrier frequency is not clinical risk. Most recessive variants require two copies for disease expression; many dominant variants show incomplete penetrance. Read this as a population fingerprint of what's in the gene pool, not a per-dog prediction.

Degenerative Myelopathy (DM)
Autosomal recessive (Incomplete penetrance)
low 0.69%
n = 9,035 dogs · 1 variant tested · OMIA:000263-9615 · omia.org →
SOD1what this gene does

SOD1 is a gene that helps protect cells from damage caused by harmful molecules called free radicals.

For your dog: If your dog is a carrier of SOD1 variants, it's worth discussing with your vet, but remember carrier status doesn't mean your dog will get the disease.

n = 9,035 dogs · 1 variant tested · OMIA:001402-9615 · omia.org →
ABCB1what this gene does

ABCB1 is a gene that helps control how certain drugs are processed and cleared from a dog's body.

For your dog: If your dog is from a breed that carries this gene variant, ask your vet about medication sensitivities before giving any new drugs.

n = 9,035 dogs · 2 variants tested · OMIA:000162-9615 · omia.org →
PDK4what this gene does

PDK4 helps regulate how cells use energy, especially in the heart muscle.

For your dog: If your dog is one of the breeds known to carry this gene, it’s worth discussing heart health with your vet, but being a carrier doesn’t mean your dog will develop disease.

n = 8,995 dogs · 1 variant tested · OMIA:000157-9615 · omia.org →
FGF4what this gene does

FGF4 influences leg length by affecting bone growth, leading to shorter legs in certain breeds.

For your dog: If your dog is from a breed known to carry this gene, it's worth discussing spinal health with your vet, but being a carrier doesn’t guarantee problems.

Collie Eye Anomaly (CEA)
Autosomal recessive
low 0.16%
n = 9,034 dogs · 1 variant tested · OMIA:000218-9615 · omia.org →
NHEJ1what this gene does

NHEJ1 is a gene involved in repairing breaks in DNA, helping maintain the integrity of genetic information in cells.

For your dog: If your dog belongs to one of the breeds known to carry this gene variant, it's worth discussing testing with your vet to understand any potential eye health risks.

n = 9,014 dogs · 1 variant tested · OMIA:001298-9615 · omia.org →
PRCDwhat this gene does

PRCD is a gene involved in the health of a dog's retina, the part of the eye that detects light and helps with vision.

For your dog: If your dog belongs to a breed known to carry PRCD changes, it's worth discussing eye health and potential genetic testing with your vet.

low <0.1%
n = 9,035 dogs · 1 variant tested · OMIA:001057-9615 · omia.org →
Cone-Rod Dystrophy (cord1-PRA/crd4)
Autosomal recessive (Incomplete penetrance)
low <0.1%
n = 9,017 dogs · 1 variant tested · OMIA:001432-9615 · omia.org →
RPGRIP1what this gene does

RPGRIP1 is a gene involved in the function of photoreceptor cells in the eye, which help dogs see in different light conditions.

For your dog: If your dog belongs to a breed known to carry RPGRIP1 mutations, it’s worth discussing with your vet to understand the risks and monitor eye health.

n = 9,035 dogs · 2 variants tested · OMIA:002120-9615 · omia.org →
NDRG1what this gene does

NDRG1 is a gene involved in nerve cell function and maintenance, helping keep the nervous system working properly.

For your dog: If your dog is from a breed known to carry NDRG1 variants, it’s worth discussing with your vet, especially if you notice any mobility issues.

low <0.1%
n = 9,035 dogs · 3 variants tested · OMIA:000256-9615 · omia.org →
SLC3A1what this gene does

SLC3A1 is a gene that helps transport certain amino acids in the kidneys. It plays a key role in preventing the buildup of cystine, which can form stones.

For your dog: If your dog is from a breed known to carry SLC3A1 variants, it’s worth discussing cystinuria risks with your vet, especially if urinary issues arise.

n = 9,034 dogs · 1 variant tested · OMIA:002179-9615 · omia.org →
ABCA4what this gene does

ABCA4 is a gene that helps manage the transport of molecules in the retina, the part of the eye responsible for vision.

For your dog: If your dog is from a breed known to carry ABCA4 variants, it's worth discussing eye health with your vet, especially as they age.

Cystinuria Type I-B (SLC7A9 p.A217T)
Autosomal recessive (Incomplete penetrance)
low <0.1%
n = 9,035 dogs · 2 variants tested · OMIA:001880-9615 · omia.org →
SLC7A9what this gene does

SLC7A9 is a gene that helps transport certain amino acids in the kidneys. It plays a role in how the body handles cystine, an amino acid that can form crystals.

For your dog: If your dog is a carrier, it’s worth discussing with your vet to monitor urinary health and catch any issues early.

n = 9,035 dogs · 1 variant tested · OMIA:002365-9615 · omia.org →
RBM20what this gene does

RBM20 is a gene that helps control how the heart muscle builds and repairs itself. It plays a key role in keeping the heart's pumping function strong.

For your dog: If you have a dog from these breeds, it’s worth discussing heart health with your vet, especially as your dog ages.

Canine Scott Syndrome (CSS)
Autosomal recessive
low <0.1%
n = 9,035 dogs · 1 variant tested · OMIA:001353-9615 · omia.org →
ANO6what this gene does

ANO6 is a gene that helps regulate how blood cells expose certain signals on their surface, which is important for normal blood clotting.

For your dog: If your dog is from a breed known to carry ANO6 mutations, it’s worth discussing with your vet before any procedures to ensure bleeding risks are managed.

n = 9,035 dogs · 2 variants tested · OMIA:001365-9615 · omia.org →
low <0.1%
n = 9,035 dogs · 1 variant tested · OMIA:001523-9615 · omia.org →
n = 9,035 dogs · 1 variant tested · OMIA:000831-9615 · omia.org →
n = 9,027 dogs · 1 variant tested · OMIA:002015-9615 · omia.org →
FAM20Cwhat this gene does

FAM20C is a gene that plays a key role in the mineralization of teeth, helping them develop properly and stay strong.

For your dog: If your dog is from one of these breeds, it's worth mentioning FAM20C to your vet when discussing dental care, but being a carrier doesn't mean your dog will have problems.

Exercise-Induced Collapse (EIC)
Autosomal recessive (Incomplete penetrance)
low <0.1%
n = 9,032 dogs · 1 variant tested · OMIA:001466-9615 · omia.org →
DNM1what this gene does

DNM1 is a gene that helps nerve cells communicate properly by managing how they send signals during muscle activity.

For your dog: If your dog belongs to one of the breeds known to carry this gene variant, it's worth discussing EIC with your vet, especially if your dog is very active or shows signs of weakness during exercise.

Plus 8 more at lower frequency. Full table available via the API when shipped.
Source: Donner J et al. 2023. Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651 · Evidence: Limited (DTC ascertainment, tag-SNP proxy) · Confounding MEDIUM · License CC-BY-4.0 · Phene IDs from OMIA (Sydney School of Veterinary Science, The University of Sydney; DOI 10.25910/2AMR-PV70).
Sample size in this breed: 9,035 dogs from the Donner 2023 cohort.

Which Mendelian variants matter most for Siberian Huskies?

The Mendelian-disease table above lists 194 variants screened in 9,035 Siberian Huskies (Donner 2023). The carrier frequencies are low across the board, well under 1% for most conditions, which reflects the breed’s relatively tight genetic structure. None reaches the threshold of widespread concern that would reshape a routine screening recommendation. Still, twelve variants appear at observable carrier frequency.

Degenerative Myelopathy (DM)

Degenerative myelopathy in Siberian Huskies is a progressive neurological disease caused by a variant in SOD1. It is a late-onset spinal-cord degeneration that begins with hind-limb weakness and progresses over months to years (Wininger et al. 2011, J Vet Intern Med 25:1014-1021). The disease is autosomal recessive with incomplete penetrance, meaning not every dog with two copies becomes symptomatic. 0.69% of Siberian Huskies in the Donner cohort carry the variant (n=9,035). That is roughly 1 in 145 dogs.

Testing is available from most commercial DNA labs. Affected dogs are managed symptomatically with physical therapy and supportive care.

MDR1 (Multidrug Resistance 1) Medication Sensitivity

MDR1 medication sensitivity in Siberian Huskies is a pharmacogenetic condition caused by a variant in the ABCB1 gene. Dogs with one or two copies have reduced drug transport across the blood-brain barrier, which makes certain medications, most notably the antiparasitic ivermectin, accumulate to toxic levels in the central nervous system. The variant is autosomal dominant. 0.68% of Siberian Huskies in the Donner cohort carry it (n=9,035).

Penetrance is incomplete; in the Donner 2023 cohort, 1 of 2 at-risk dogs was phenotype-confirmed, placing an upper bound of 50% on penetrance in this dataset. Testing is widely available. Affected dogs simply avoid ivermectin-based dewormers and heartworm preventives, the breed typically switches to milbemycin or fenbendazole instead.

How should I test my Siberian Husky?

Given the low carrier frequencies across the Mendelian panel, a breed-specific screening panel is optional rather than critical. If you are a breeder, a panel covering DM, MDR1, and the two retinal dystrophies (PRCD and CORD1) is the reasonable starting point. For a pet-only dog, testing is less urgent unless there is family history for a specific condition.

What should I feed a Siberian Husky?

Siberian Huskies were bred for distance and cold, and their metabolism still expects work. A pet Husky eating maintenance kibble in a temperate house is being fed for a job they aren’t doing. Weight management becomes the primary nutritional challenge.

Joint care should follow large-breed guidelines even though Huskies sit at the upper end of the medium-size range. Adult Huskies weigh 45 to 60 pounds. Growth from puppy to adult is moderately rapid but not the extreme scaling of giant breeds. A large-breed puppy formulation with calcium-to-phosphorus ratio between 1.1:1 and 2:1 aligns with NRC 2006 recommendations. Monitor body condition monthly during growth; the breed’s working heritage can mask overfeeding until adulthood hits.

The working-dog metabolism demands high-quality protein and fat. Huskies thrive on formulations with 25% to 30% protein and 15% to 20% fat as fed. The breed does well on both grain-inclusive and grain-free diets; no breed-specific DCM signal has been reported for Siberian Huskies in the FDA’s 2018 and 2022 grain-free diet advisories (FDA 2018, FDA 2022). The choice between the two depends on individual dog response. Watch for coat quality and energy level as indicators of whether a diet is matching the breed’s high metabolic expectations.

Weight gain is the most common nutritional failure in pet Huskies. A working sled dog can burn over 10,000 kcal per day in racing conditions (Hinchcliff et al. 1997, J Nutr 127:2494S-2502S). A pet Husky in a suburban yard needs a fraction of that, and portion control is essential to prevent weight gain. Free-feeding is a near-guarantee of obesity by age three. Measure portions by the breed-club standard: feed to body condition, not to the bowl recommendation on the bag.

What we don’t know

The Siberian Husky atlas is built from 30 dogs across 2 sub-populations. That is a small foundation for breed-wide inference. Carrier frequencies are reliable for the variants reported, but the breed’s true genetic structure, whether there are undetected sub-populations with different allele frequencies, whether rare variants exist in field lines that didn’t make it into the sequencing cohort, remains partly opaque. A larger, geographically diverse atlas would refine those estimates.

The interaction between MDR1 carrier status and actual ivermectin sensitivity in Huskies specifically is incompletely studied. The MDR1/ABCB1 research base is strongest in Collies, but the variant mechanism is consistent across breeds. A Husky with a positive test should avoid ivermectin-class drugs; breed-specific penetrance data from Husky field populations has not been published to date.

Frequently asked questions about Siberian Huskies

What is the most common genetic disease in Siberian Huskies? Degenerative myelopathy is the most frequent Mendelian condition by carrier frequency, with 0.69% of the breed carrying the variant (Donner 2023, n=9,035). Affected dogs develop progressive hind-limb weakness, typically in later life (Wininger et al. 2011, J Vet Intern Med 25:1014-1021). Most conditions in Huskies are well below 1% carrier frequency.

Should I do a DNA test on my Siberian Husky? For breeding stock, a panel covering DM and the two retinal dystrophies (PRCD and CORD1) is reasonable. For a pet dog, testing is optional unless there is family history for a specific condition or you are planning future litters.

Are Siberian Huskies prone to weight gain? Yes. The breed was selected for distance work in extreme cold and has a metabolism that expects that job. Pet Huskies in temperate climates gain weight easily on standard portions. Measured feeding and regular body-condition scoring are essential.

How long do Siberian Huskies live? The atlas-derived median lifespan is 11.9 years. Individual dogs vary; the 11.9-year atlas median reflects the full population, and well-managed dogs may exceed it.

What is the best diet for a Siberian Husky? High-protein (25-30%), moderate-to-high fat (15-20% as fed) formulations that match the breed’s working heritage. Both grain-inclusive and grain-free diets work well; no breed-specific DCM signal has been reported for Siberian Huskies in the FDA 2018 and 2022 grain-free diet advisories (FDA 2018, FDA 2022). Feed to body condition, not appetite. Free-feeding without portion control is strongly associated with obesity in low-activity dogs (NRC 2006).

Are Siberian Huskies good with kids? Siberian Huskies have high prey drive but low human-directed aggression. They are commonly kept in families with children. Supervision around small children is standard practice for any breed this size. Prey-drive training toward other small animals (cats, rabbits) should start early.

What is the most common health problem in Siberian Huskies? Obesity and joint stress from excess weight are the most common nutritional and musculoskeletal issues. Hip dysplasia and other joint disease are breed concerns but not dominant. Genetic disease is rare at the Mendelian level.

Do Siberian Huskies need a lot of exercise? Yes. The breed was selected for all-day work in snow. A pet Husky needs a minimum of one to two hours of vigorous aerobic activity daily. Under-exercised Huskies develop destructive behavior and weight gain. High-prey-drive play (fetch, spring-pole work, lure coursing) is better suited to the breed than treadmill walking.

A gift to human medicine

Siberian Huskys are a natural model for human disease

Because the same genes cause the same conditions across species, the inherited conditions documented in Siberian Huskys help researchers understand, and work toward treating, the human diseases they model. This is the dog advancing human medicine. The breed models the human disease; it does not have it, and this is not a prediction for your dog.

Human equivalents via OMIA → Mondo / OMIM. Model-of, not identity.
Documented in OMIA

Every condition recorded in the Siberian Husky

Beyond the testable carriers above, OMIA's literature catalogue records 12 genetic conditions in the Siberian Husky, 10 of which have a known human equivalent. This is the documented landscape across all Siberian Huskys ever studied, not a prediction for any one dog.

Online Mendelian Inheritance in Animals (OMIA); Nicholas, Tammen & Sydney Informatics Hub, DOI 10.25910/2AMR-PV70
Documented in the breed's literature is not carrier status and not a forecast for an individual dog. Human equivalents are mapped via Mondo/OMIM. Carrier frequencies (above) are the separately-measured testable subset (Donner 2023).
The data behind this page

Where every number on this page came from.

This page draws on three primary data sources. Carrier frequencies for the Mendelian section come from Donner et al. 2023 (CC-BY-4.0). We grade these data at evidence Limited because the cohort is a direct-to-consumer ascertainment, which biases toward owners who chose to test their dogs. The panel also uses tag-SNP proxies for some variants rather than direct causal-variant assays. Limited is a study-design grade, not a quality grade: the Donner cohort is the largest open canine-genotype dataset in existence and we are grateful for it. We rate the confounding MEDIUM.

Population-genetic dimensions (heterozygosity, intra-breed PCA distance, nearest neighbors, trait-locus frequencies) come from CanVAS (Brundage 2026), harmonized through the Sniff Atlas. The exact release date and verification commit are pinned at the bottom of the page so a researcher can trace a number back to a specific snapshot. The disease-gene-variant graph comes from OMIA (Online Mendelian Inheritance in Animals; Nicholas, Tammen, and the Sydney Informatics Hub at the Sydney School of Veterinary Science, The University of Sydney; retrieved April 2026, DOI 10.25910/2AMR-PV70).

What this page does not yet have. Inheritance modes and per-disease penetrance evidence from Donner 2023 are now in the structured data for every variant the panel covers. Mondo, OMIM, Ensembl, and HGNC cross-references on gene pages remain pending, they arrive in December 2026 alongside the imputed 9.67M-variant CanVAS dataset via the OMIA SQL dump absorption. Until then, gene IDs carry NCBI Gene and OMIA phene URLs only; the wider human-homolog and disease-ontology cross-reference set fills in with that release.

How to cite this page. The computed dimensions on this page are derived from the open Sniff Atlas v1.0.1 (Gehring 2026, doi:10.5281/zenodo.20566358, CC-BY 4.0). Full citation formats including BibTeX, RIS, and CITATION.cff at sniff.world/cite.

Add your siberian husky to the atlas

We have 30 siberian huskys. We do not have yours.

Every siberian husky added sharpens the breed's genetic neighborhood. Enrollment is free. The data stays open. The star is permanent.

Want to wait for DNA uploads?

Leave your email and we'll let you know the moment DNA uploads open for Siberian Huskys.

References
  1. Donner J, Freyer J, Davison S, Anderson H, Blades M, Honkanen L, et al. (2023). Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651. doi:10.1371/journal.pgen.1010651
  2. Brundage J, et al. (2026). CanVAS: a harmonized canine variant atlas. bioRxiv. doi:10.64898/2026.04.13.718238
  3. Nicholas, F.W., Tammen, I., & Sydney Informatics Hub. (2026). Online Mendelian Inheritance in Animals (OMIA) [dataset]. The University of Sydney. https://omia.org. doi:10.25910/2AMR-PV70 (retrieved April 2026).
Last updated
Sources: CanVAS (Brundage 2026) · Donner 2023 · OMIA