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Soft Coated Wheaten Terrier

Soft Coated Wheaten Terrier
Photo: Terrierkännaren / CC BY-SA 3.0 · Wikimedia

16 Soft Coated Wheaten Terriers in the atlas. Every number on this page has a source.

Population-genetic snapshot of Soft Coated Wheaten Terriers in the Sniff Atlas, source-graded Mendelian carrier frequencies from Donner 2023, and nutrition guidance tied to the genetic findings above.

Also known as Wheaten and Wheatie.

See in the atlas → From CanVAS
The plain version

Soft Coated Wheaten Terriers have a moderately diverse genetic background, which is a good sign for their overall health. They share some traits with breeds like the Airedale and Border Terriers. While we don’t have specific details on their size or lifespan here, these dogs are known for their friendly nature and soft, wavy coats. Some health issues, like Degenerative Myelopathy and certain kidney conditions, have been found in the breed’s gene pool, so it’s a good idea to talk with your vet or consider genetic testing to keep your dog happy and healthy. Since this info comes from a small group of dogs, it’s a helpful starting point but may not cover every detail.

What the atlas says about Soft Coated Wheaten Terrier

In the atlas, the Soft Coated Wheaten Terrier clusters consistently as Soft Coated Wheaten Terrier (100% of the 16 dogs here). At the trait loci, RSPO2 runs higher than the atlas average (100% here vs 55%); ADAMTS17 runs higher than the atlas average (96% here vs 54%). Dogs here sit in a relatively sparse region of the atlas, fewer close neighbors than typical.

Only 16 dogs of this breed in the atlas, modestly sampled.

Genetic dimensions · CanVAS atlas

What the genome says about Soft Coated Wheaten Terrier

Computed from the 18,477 research dogs in the Atlas.

These figures are computed from only 16 Soft Coated Wheaten Terriers in the atlas. Treat them as provisional. They sharpen as more dogs are added.
Dogs in the Atlas
16Founders
10 from Hayward2016, 4 from Spatola, 2 from JenkinsWGS
Genetic diversity

Not enough dogs in the atlas yet (n=16) for a reliable diversity figure. It fills in as more are added.

Mean heterozygosity across the breed. Too few dogs in this breed (<20) to rank.
What does genetic diversity mean?

How varied a breed's gene pool is — the share of gene spots where a typical dog of the breed carries two different versions rather than two identical ones.

How to read it: Higher = more diverse. Among well-sampled breeds it ranges roughly 0.22 (least diverse) to 0.33 (most diverse).

Diversity is a strength, not a verdict on any individual dog. Lower diversity means it's worth paying attention to recessive-risk testing — not that a dog is doomed.

Cluster structure

Not enough dogs in the atlas yet (n=16) to resolve cluster structure. It fills in as more are added.

What does within-breed variation mean?

How much individual dogs within the breed differ from each other genetically.

How to read it: Higher = more internal variety among individuals of the breed.

Sensitive to how many dogs of the breed we've sampled.

Related breeds
In the Terrier group
Explore the full lineage map →
VBO foundation stock (breeding records) · AKC breed group
Relatedness is documented lineage + kennel family. Genetic-ancestry distance measures diversity, not kinship, so it isn't used here.
How long they live
13.7years (life expectancy)
95% CI 13.2–14.4 · VetCompass, McMillan 2024, n=620. source
What does typical lifespan mean?

The median age dogs of the breed tend to reach.

How to read it: Higher = longer-lived. Compare to longevity-for-size to see whether it's just a size effect.

Drawn from population lifespan records; individual dogs vary widely with care, genetics, and luck.

Trait genetics
Allele frequencies at named morphology loci

Frequency of the alternate allele in this breed at each locus's representative SNP.

Not enough Soft Coated Wheaten Terriers in the atlas yet (n=16) for reliable allele frequencies at these loci. It fills in as more are added.

n = 16 dogs · low confidence · CanVAS (Brundage 2026) · Sniff Atlas
Names & origins

Other names

The Soft Coated Wheaten Terrier is also recorded as Wheaten and Wheatie.

Identified as Soft Coated Wheaten Terrier (VBO:0201260) in the Vertebrate Breed Ontology (Mullen et al. 2025, CC-BY 4.0) · registry IDs iDog 228 · VeNom 22411.

What you see when you look at a Soft Coated Wheaten Terrier

What does the genome say about how a Soft Coated Wheaten Terrier looks?

Soft Coated Wheaten Terriers look the way they do because of a small set of fixed and near-fixed morphology genes that, taken together, define the visible breed. Each translation below pairs the gene with the trait an owner actually sees, the breed's allele frequency at that locus, and a one-clause causal phrase.

Where the breed-defining genes act, mapped on a generic dog-body key — and how fixed each marker is in the Soft Coated Wheaten Terrier. The figure is the most-settled marker we read in that region; the full per-locus panel is below. (The silhouette is a shared anatomical guide, not this breed's outline.)

Body sizeLCORL · 100%Skull shapeSMOC2 · 100%EarsMSRB3 · 100%Leg lengthFGF4 CFA18 · 100%Coat & colorRSPO2 · 100%
CanVAS trait-locus panel (Brundage 2026)
15 morphology markers read across 5 regions. Allele frequency = how fixed a marker is in this breed, not whether your dog carries it.

Size and build

IGF1 sits at 75% for the small-body allele. IGF1 is the gene that sets dog body size from Chihuahua to Great Dane. Intermediate frequencies typically keep a breed in the mid-sized range rather than tipping toward the larger working forms.

IGF1what this gene does

IGF1 is a gene that plays a key role in determining a dog's body size. It influences how much a dog grows, affecting overall stature.

For your dog: Knowing about IGF1 gives you insight into your dog's size traits, but it’s just one part of the bigger picture when it comes to their health and care.

Full IGF1 gene page →

HMGA2 is at 28%, leaving most of the size signal to other loci in the panel.

HMGA2what this gene does

HMGA2 is a gene that influences body size in dogs, helping determine how big or small a dog grows.

For your dog: Knowing about HMGA2 helps you appreciate the genetic factors behind your dog's size, but it doesn't signal any health issues.

Full HMGA2 gene page →

SMAD2 is near-fixed at 94%, a chromosome-7 height locus differentiating small from giant breeds.

SMAD2what this gene does

SMAD2 is a gene involved in regulating body size by influencing how cells grow and develop.

For your dog: Knowing about SMAD2 helps understand your dog's size traits but isn't linked to health issues; no immediate action needed.

Full SMAD2 gene page →

LCORL is near-fixed at 100%, the NCAPG/LCORL height locus that is one of the strongest single contributors to canine body size.

LCORLwhat this gene does

LCORL is a gene that influences body size in dogs. It helps determine how big or small a dog might grow.

For your dog: Knowing about LCORL helps you appreciate the genetic factors behind your dog's size, but it’s just one piece of the bigger picture when it comes to health and care.

Full LCORL gene page →

STC2 is near-fixed at 100%, modulating growth-axis signaling toward the breed's body-size set point.

ADAMTS17 is at 96%, near-fixed for the size variant.

ADAMTS17what this gene does

ADAMTS17 is a gene that influences body size and also plays a role in certain eye conditions. It affects the structure of tissues in the eye and elsewhere in the body.

For your dog: If your dog belongs to a breed known to carry ADAMTS17 variants, it’s worth discussing genetic testing and eye exams with your vet to stay ahead of potential issues.

Full ADAMTS17 gene page →

Leg length

The FGF4 retrogene on chromosome 18 is near-fixed in this breed at 100%. This is the leg-length variant. The breed is fully committed to the long-legged form rather than the short-legged Corgi-and-Dachshund body plan.

The FGF4 retrogene on chromosome 12 is near-fixed at 100%, the chondrodystrophic variant associated with intervertebral disc disease risk in breeds that carry it.

Coat type, length, and color

RSPO2 is near-fixed at 100% for the furnishings allele, the genetic basis of the eyebrows-and-mustache pattern seen in Schnauzers and Wheaten Terriers.

RSPO2what this gene does

RSPO2 influences the texture and appearance of a dog's coat, particularly the presence of 'furnishings' like mustaches and eyebrows. It helps determine whether a dog has that distinctive wiry or textured look.

For your dog: If your dog has those wiry eyebrows or a mustache, RSPO2 is part of the reason—no health worries, just a coat feature worth knowing about.

Full RSPO2 gene page →

FGF5 sits at 69% for the long-coat variant. Coat length is influenced by other loci as well, so intermediate FGF5 frequencies do not always correspond to intermediate visible coat lengths.

FGF5what this gene does

FGF5 is a gene that influences the length of a dog's coat. It acts like a natural switch, telling hair follicles when to stop growing longer fur.

For your dog: If your dog has a notably long or short coat, FGF5 is likely part of the reason—no action needed, but it’s a neat genetic detail to know.

Full FGF5 gene page →

KRT71 is near-fixed at 100% for the wavy/curly variant. Coat curl phenotype varies across breeds at this fixation depending on modifier loci, and visible expression is not always curled even when the locus is fixed.

KRT71what this gene does

KRT71 is a gene that influences the curliness of a dog's coat. It helps determine whether a dog's fur is straight or has a distinctive curl.

For your dog: If your dog has a curly coat, KRT71 is likely part of the reason; it’s a natural variation, not a health concern.

Full KRT71 gene page →

MC1R sits at 81% at the representative SNP. MC1R controls the switch between red-to-gold pigment and black-to-brown pigment, with the e/e homozygous genotype producing the gold-to-red spectrum. Substrate frequencies at this SNP depend on the array's polarity, so visible coat color in the breed is a more reliable indicator than this single number.

MC1Rwhat this gene does

MC1R is a gene that influences coat color in dogs, affecting how pigments are produced in the fur.

For your dog: Knowing about MC1R gives insight into your dog's coat color but doesn't relate to health issues.

Full MC1R gene page →

Ears

MSRB3 is at 100% for the drop-ear allele, the genetic basis of the breed's signature dropped ear set.

MSRB3what this gene does

MSRB3 is a gene involved in the development of ear shape and structure in dogs.

For your dog: Understanding MSRB3 helps explain why your dog's ears look the way they do, but it isn't linked to any health issues.

Full MSRB3 gene page →

Skull shape

BMP3 sits at 65%, contributing to the breed's moderate, mesaticephalic head shape rather than the extreme brachycephalic form.

BMP3what this gene does

BMP3 is a gene that influences the shape of a dog's skull, particularly contributing to a shorter, broader head shape known as brachycephaly.

For your dog: If your dog has a broad, short skull, it's worth discussing with your vet how this might impact their health, even though BMP3 isn't directly tied to illness.

Full BMP3 gene page →

SMOC2 is at 100%, the major locus contributing to the breed's brachycephalic face shape.

SMOC2what this gene does

SMOC2 influences the shape of a dog's skull, particularly affecting how flat or short the face appears.

For your dog: If your dog has a short nose, it's worth discussing with your vet how this trait might impact their health over time.

Full SMOC2 gene page →
Mendelian-disease genetics

What genetic diseases do Soft Coated Wheaten Terriers carry?

From a panel of 250 Mendelian-disease variants screened in 1,054,293 dogs (Donner et al. 2023), Soft Coated Wheaten Terriers carry 8 of them at observable frequency. Carrier frequency is not clinical risk. Most recessive variants require two copies for disease expression; many dominant variants show incomplete penetrance. Read this as a population fingerprint of what's in the gene pool, not a per-dog prediction.

Degenerative Myelopathy (DM)
Autosomal recessive (Incomplete penetrance)
high 21.1%
n = 607 dogs · 1 variant tested · OMIA:000263-9615 · omia.org →
SOD1what this gene does

SOD1 is a gene that helps protect cells from damage caused by harmful molecules called free radicals.

For your dog: If your dog is a carrier of SOD1 variants, it's worth discussing with your vet, but remember carrier status doesn't mean your dog will get the disease.

Protein Losing Nephropathy (PLN; NPHS1-related)
Autosomal recessive (Incomplete penetrance)
moderate 18.0%
n = 607 dogs · 1 variant tested · OMIA:001326-9615 · omia.org →
NPHS1what this gene does

NPHS1 is a gene important for kidney function, helping to keep proteins in the blood where they belong.

For your dog: If your dog’s breed is on the list, it’s worth asking your vet about NPHS1 to understand any risks and keep an eye on kidney health.

Cystinuria Type I-B (SLC7A9 p.A217T)
Autosomal recessive (Incomplete penetrance)
low 6.8%
n = 607 dogs · 2 variants tested · OMIA:001880-9615 · omia.org →
SLC7A9what this gene does

SLC7A9 is a gene that helps transport certain amino acids in the kidneys. It plays a role in how the body handles cystine, an amino acid that can form crystals.

For your dog: If your dog is a carrier, it’s worth discussing with your vet to monitor urinary health and catch any issues early.

Microphthalmia (Discovered in the Soft-Coated Wheaten Terrier)
Autosomal recessive
low 3.8%
n = 607 dogs · 1 variant tested · OMIA:002151-9615 · omia.org →
Paroxysmal Dyskinesia (Discovered in the Irish Soft Coated Wheaten Terrier; PIGN-related; PxD)
Autosomal recessive
low 3.1%
n = 606 dogs · 1 variant tested · OMIA:002084-9615 · omia.org →
Cystinuria Type I-A (SLC3A1 p.I192V)
Autosomal recessive
low 0.49%
n = 607 dogs · 3 variants tested · OMIA:000256-9615 · omia.org →
SLC3A1what this gene does

SLC3A1 is a gene that helps transport certain amino acids in the kidneys. It plays a key role in preventing the buildup of cystine, which can form stones.

For your dog: If your dog is from a breed known to carry SLC3A1 variants, it’s worth discussing cystinuria risks with your vet, especially if urinary issues arise.

Cone-Rod Dystrophy (cord1-PRA/crd4)
Autosomal recessive (Incomplete penetrance)
low 0.41%
n = 605 dogs · 1 variant tested · OMIA:001432-9615 · omia.org →
RPGRIP1what this gene does

RPGRIP1 is a gene involved in the function of photoreceptor cells in the eye, which help dogs see in different light conditions.

For your dog: If your dog belongs to a breed known to carry RPGRIP1 mutations, it’s worth discussing with your vet to understand the risks and monitor eye health.

Progressive Rod-Cone Degeneration (prcd-PRA)
Autosomal recessive
low <0.1%
n = 605 dogs · 1 variant tested · OMIA:001298-9615 · omia.org →
PRCDwhat this gene does

PRCD is a gene involved in the health of a dog's retina, the part of the eye that detects light and helps with vision.

For your dog: If your dog belongs to a breed known to carry PRCD changes, it's worth discussing eye health and potential genetic testing with your vet.

Source: Donner J et al. 2023. Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651 · Evidence: Limited (DTC ascertainment, tag-SNP proxy) · Confounding MEDIUM · License CC-BY-4.0 · Phene IDs from OMIA (Sydney School of Veterinary Science, The University of Sydney; DOI 10.25910/2AMR-PV70).
Sample size in this breed: 607 dogs from the Donner 2023 cohort.

Which Mendelian variants matter most for Soft Coated Wheaten Terriers?

The Mendelian-disease table above lists 8 variants at observable carrier frequency among 194 screened in 607 Soft Coated Wheaten Terriers (Donner 2023). Two matter most by frequency and impact; four others are worth knowing.

Degenerative Myelopathy (DM)

Degenerative myelopathy in Soft Coated Wheaten Terriers is a progressive spinal-cord degeneration caused by a recessive variant in SOD1. Affected dogs lose hind-limb function over months to years, typically emerging in middle age. The condition is incurable and eventually fatal. 21.1% of Wheaten Terriers in the Donner cohort carry the variant (n=607). That is roughly one in five.

The incomplete penetrance means not every dog with two copies becomes clinically affected. Testing is available. The Soft Coated Wheaten Terrier Club of America includes DM screening in their health recommendations for breeding stock.

Protein losing nephropathy in Soft Coated Wheaten Terriers is a kidney disease caused by a recessive NPHS1 variant. Affected dogs lose protein in the urine, leading to edema, ascites, and progressive renal failure. 18.0% of Wheaten Terriers in the Donner cohort carry the variant (n=607).

Incomplete penetrance means not every carrier pair produces a clinically affected dog. Diagnosis is by urinalysis and kidney imaging. Affected dogs are managed with diet (reduced protein, low sodium) and monitoring for progression. Testing is available.

Cystinuria Type I-B (SLC7A9 p.A217T)

Cystinuria Type I-B in Soft Coated Wheaten Terriers is caused by an SLC7A9 variant that leads to excess urinary cystine excretion and bladder-stone formation. 6.8% of Wheaten Terriers carry the variant (n=607).

Not every dog with two copies forms stones. Affected dogs are managed with diet (low-protein, alkalinizing) and increased water intake. Testing is available.

Microphthalmia and Paroxysmal Dyskinesia

Microphthalmia in Soft Coated Wheaten Terriers is a recessive eye condition causing underdevelopment of one or both eyes. 3.8% carry the variant (n=607). Paroxysmal dyskinesia is a recessive neuromuscular condition causing episodes of involuntary movement or stiffness. 3.1% carry the variant (n=606). Both are rare in homozygous state. Testing is available for breeding decisions.

How should I test my Soft Coated Wheaten Terrier?

A breed-specific panel covering DM, PLN, and both cystinuria types (SLC7A9 and SLC3A1) is the high-yield starting point. Additional testing for microphthalmia, paroxysmal dyskinesia, and the retinal-dystrophy variants is justified for breeding stock. Any CLIA-accredited canine genetics lab can run these panels.

What should I feed a Soft Coated Wheaten Terrier?

Feeding a Wheaten Terrier well means accounting for the breed’s protein-losing nephropathy risk and managing weight appropriately for a compact, muscular terrier. The 18% carrier frequency for NPHS1-related PLN shapes the nutritional priorities most directly.

Protein management is breed-specific. A healthy Wheaten Terrier without clinical kidney disease should eat a moderate-protein (18 to 22% on a dry-matter basis) complete and balanced adult formula. The breed is prone to weight gain if overfed, which compounds kidney stress. Research on NPHS1-related PLN in Soft Coated Wheaten Terriers has not yet identified a preventive diet, but protein restriction is standard practice once clinical disease emerges.

Joint care is straightforward because hip dysplasia is not a major breed concern. Wheaten Terriers are a medium-sized breed not documented among the high-prevalence hip dysplasia breeds in OFA statistics (ofa.org/diseases/hip-dysplasia/statistics/) and do not require the specialized puppy formulations that giant breeds do. A standard large-breed puppy formula with controlled calcium (0.8 to 1.6% on a dry-matter basis) is appropriate during growth.

Feeding schedules matter for metabolic health. Wheaten Terriers are food-motivated and prone to obesity. Two meals per day on a measured portion appropriate to activity level is the standard. Unlike scent hounds bred for long-range hunting, Wheaten Terriers are compact terriers with moderate energy demands. A typical adult Wheaten in the 30 to 45 lb range needs roughly 900 to 1,200 kilocalories per day at maintenance, depending on age and activity level (NRC 2006). Weight trending upward is the first sign to reduce portion size.

Grain-free diets carry no specific signal in this breed. Wheaten Terriers are not among the breeds over-represented in FDA reports of diet-associated dilated cardiomyopathy (FDA 2019 advisory, fda.gov). A grain-inclusive or grain-free formula is acceptable if it is complete and balanced, taurine-supplemented, and tested in feeding trials by the manufacturer.

What we don’t know

The penetrance of both DM and PLN in Soft Coated Wheaten Terriers remains incompletely characterized. We do not yet know which dogs carrying the DM variant (21.1% of the cohort) or the PLN variant (18.0% of the cohort) will develop clinical signs and which will remain asymptomatic for life. The breed’s small atlas sample (n=16 dogs) limits our ability to identify longevity outliers or environmental modifiers that might inform that distinction.

Environmental and dietary factors in PLN progression have not been systematically studied in Wheaten Terriers specifically. The honest summary is that clinical management is based on general kidney-disease principles rather than breed-specific evidence.

Frequently asked questions about Soft Coated Wheaten Terriers

What is the most common genetic disease in Soft Coated Wheaten Terriers? Degenerative myelopathy (DM), with 21.1% carrier frequency in the Donner cohort (Donner 2023, n=607). Protein losing nephropathy is close behind at 18.0% carrier frequency (Donner 2023, n=607). Both are inherited as autosomal recessives with incomplete penetrance.

Are Soft Coated Wheaten Terriers prone to kidney disease? Protein losing nephropathy is a breed concern, with 18.0% of Wheaten Terriers carrying the NPHS1 variant (Donner 2023, n=607). Not every carrier pair produces an affected dog, but screening breeding stock is recommended by the breed club.

What is degenerative myelopathy and when does it show up? Degenerative myelopathy is a progressive spinal-cord disease that causes hind-limb weakness and eventually paralysis. It typically emerges in middle age to senior years. The condition is incurable. Genetic testing can identify carriers.

Should I do a DNA test on my Soft Coated Wheaten Terrier? For breeding stock, yes. The Soft Coated Wheaten Terrier Club of America recommends testing for DM, PLN, and cystinuria variants before breeding. For pet dogs, testing informs you of carrier status but does not change management unless clinical signs emerge.

How long do Soft Coated Wheaten Terriers live? The breed median lifespan is commonly cited as approximately 12 to 14 years by the Soft Coated Wheaten Terrier Club of America (scwtca.org). Individual longevity varies with genetics, early-life care, weight management, and screening for breed-typical conditions like kidney disease.

What should I feed a Soft Coated Wheaten Terrier? A moderate-protein (18 to 22% dry-matter basis), complete-and-balanced adult formula is appropriate for healthy adults. Portion control is essential because the breed is food-motivated and prone to weight gain. Dogs with clinical kidney disease should transition to a lower-protein, low-sodium therapeutic diet under veterinary guidance.

Are Soft Coated Wheaten Terriers good with children? Yes. The breed was developed as a working farm terrier and is known for loyalty to family. Supervision with very small children is standard practice, as with any terrier.

Do Soft Coated Wheaten Terriers have ear infections? Ear infections are not documented as a major breed concern. Ear infections are not documented as a major breed concern for Soft Coated Wheaten Terriers, and the breed is not among those flagged in OFA health surveys for chronic otitis.

A gift to human medicine

Soft Coated Wheaten Terriers are a natural model for human disease

Because the same genes cause the same conditions across species, the inherited conditions documented in Soft Coated Wheaten Terriers help researchers understand, and work toward treating, the human diseases they model. This is the dog advancing human medicine. The breed models the human disease; it does not have it, and this is not a prediction for your dog.

Human equivalents via OMIA → Mondo / OMIM. Model-of, not identity.
Documented in OMIA

Every condition recorded in the Soft Coated Wheaten Terrier

Beyond the testable carriers above, OMIA's literature catalogue records 5 genetic conditions in the Soft Coated Wheaten Terrier, 4 of which have a known human equivalent. This is the documented landscape across all Soft Coated Wheaten Terriers ever studied, not a prediction for any one dog.

Online Mendelian Inheritance in Animals (OMIA); Nicholas, Tammen & Sydney Informatics Hub, DOI 10.25910/2AMR-PV70
Documented in the breed's literature is not carrier status and not a forecast for an individual dog. Human equivalents are mapped via Mondo/OMIM. Carrier frequencies (above) are the separately-measured testable subset (Donner 2023).
The data behind this page

Where every number on this page came from.

This page draws on three primary data sources. Carrier frequencies for the Mendelian section come from Donner et al. 2023 (CC-BY-4.0). We grade these data at evidence Limited because the cohort is a direct-to-consumer ascertainment, which biases toward owners who chose to test their dogs. The panel also uses tag-SNP proxies for some variants rather than direct causal-variant assays. Limited is a study-design grade, not a quality grade: the Donner cohort is the largest open canine-genotype dataset in existence and we are grateful for it. We rate the confounding MEDIUM.

Population-genetic dimensions (heterozygosity, intra-breed PCA distance, nearest neighbors, trait-locus frequencies) come from CanVAS (Brundage 2026), harmonized through the Sniff Atlas. The exact release date and verification commit are pinned at the bottom of the page so a researcher can trace a number back to a specific snapshot. The disease-gene-variant graph comes from OMIA (Online Mendelian Inheritance in Animals; Nicholas, Tammen, and the Sydney Informatics Hub at the Sydney School of Veterinary Science, The University of Sydney; retrieved April 2026, DOI 10.25910/2AMR-PV70).

What this page does not yet have. Inheritance modes and per-disease penetrance evidence from Donner 2023 are now in the structured data for every variant the panel covers. Mondo, OMIM, Ensembl, and HGNC cross-references on gene pages remain pending, they arrive in December 2026 alongside the imputed 9.67M-variant CanVAS dataset via the OMIA SQL dump absorption. Until then, gene IDs carry NCBI Gene and OMIA phene URLs only; the wider human-homolog and disease-ontology cross-reference set fills in with that release.

How to cite this page. The computed dimensions on this page are derived from the open Sniff Atlas v1.0.1 (Gehring 2026, doi:10.5281/zenodo.20566358, CC-BY 4.0). Full citation formats including BibTeX, RIS, and CITATION.cff at sniff.world/cite.

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References
  1. Donner J, Freyer J, Davison S, Anderson H, Blades M, Honkanen L, et al. (2023). Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651. doi:10.1371/journal.pgen.1010651
  2. Brundage J, et al. (2026). CanVAS: a harmonized canine variant atlas. bioRxiv. doi:10.64898/2026.04.13.718238
  3. Nicholas, F.W., Tammen, I., & Sydney Informatics Hub. (2026). Online Mendelian Inheritance in Animals (OMIA) [dataset]. The University of Sydney. https://omia.org. doi:10.25910/2AMR-PV70 (retrieved April 2026).
Last updated
Sources: CanVAS (Brundage 2026) · Donner 2023 · OMIA