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Vizsla

Lifespan
13.5 years
95% CI 13.2 to 13.9 · McMillan 2024, n=2,965
Most carried variant
Exercise-Induced Collapse
1.6% of 318 tested · Donner 2023
Behind this page
100 dogs
in the atlas · 318 tested for variants

Vizslas have a cited life expectancy of 13.5 years (McMillan 2024 / VetCompass, n=2,965, 95% CI 13.2 to 13.9). The most carried tested variant is Exercise-Induced Collapse, in 1.6% of 318 Vizslas tested (Donner 2023).

Vizsla
Photo: Pleple2000 · CC BY-SA 3.0 · Wikimedia

Also known as Hungarian Pointer, Hungarian Short-Haired Pointer (Vizsla), Hungarian Vizla, and Hungarian Vizsla.

Mendelian-disease genetics

What genetic diseases do Vizslas carry?

From a panel of 250 Mendelian-disease variants screened in 1,054,293 dogs (Donner et al. 2023), Vizslas carry 3 of them at observable frequency. Carrier frequency is not clinical risk. Most recessive variants require two copies for disease expression; many dominant variants show incomplete penetrance. Read this as a population fingerprint of what's in the gene pool, not a per-dog prediction.

marker disputed 1.6%
n = 318 dogs · 2 variants tested · OMIA:000162-9615 · omia.org →
PDK4what this gene does

PDK4 helps regulate how cells use energy, especially in the heart muscle.

For your dog: If your dog is one of the breeds known to carry this gene, it’s worth discussing heart health with your vet, but being a carrier doesn’t mean your dog will develop disease.

Exercise-Induced Collapse (EIC)
Autosomal recessive (Incomplete penetrance)
low 1.6%
n = 318 dogs · 1 variant tested · OMIA:001466-9615 · omia.org →
DNM1what this gene does

DNM1 is a gene that helps nerve cells communicate properly by managing how they send signals during muscle activity.

For your dog: If your dog belongs to one of the breeds known to carry this gene variant, it's worth discussing EIC with your vet, especially if your dog is very active or shows signs of weakness during exercise.

Factor VII Deficiency
Autosomal recessive
low 0.94%
n = 318 dogs · 1 variant tested · OMIA:000361-9615 · omia.org →
F7what this gene does

The F7 gene helps produce a protein important for blood clotting, which stops bleeding when dogs get injured.

For your dog: If your dog is from a breed known to carry F7 variants, it's worth mentioning to your vet before any surgery or if you notice unusual bleeding.

Source: Donner J et al. 2023. Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651 · Evidence: Limited (DTC ascertainment, tag-SNP proxy) · Confounding MEDIUM · License CC-BY-4.0 · Phene IDs from OMIA (Sydney School of Veterinary Science, The University of Sydney; DOI 10.25910/2AMR-PV70).
Sample size in this breed: 318 dogs from the Donner 2023 cohort.
In plain terms

What these results mean for a Vizsla

1.6% of Vizslas in the Donner 2023 cohort carried it (n=318).

  • This is a marker, not an established cause. The curated record says a test based on it is not a good indicator of heart disease risk even in Dobermans, the breed it was found in.
    OMIA 000162-9615

    “it is evident that any DNA test based on this marker will not be a good indicator of liability to this disorder in Dobermans” OMIA 000162-9615

What to do
A carrier result for this marker is not, on its own, a reason for heart screening. Heart screening is a conversation with your veterinarian about the breed and the dog.

No test, or a mix
What this marker means in breeds other than the Doberman is not established in the sources this page draws on.

Which Mendelian variants matter most for Vizslas?

The Mendelian-disease table above lists variants screened in 318 Vizslas (Donner 2023). Three matter most, and all three are rare in the breed. The rarity is itself important information.

Exercise-Induced Collapse (EIC)

Exercise-induced collapse in Vizslas is an autosomal-recessive-with-incomplete-penetrance disorder of muscle metabolism. Affected dogs experience hind-limb weakness or collapse during or immediately after intense exercise, particularly in warm conditions. The variant is rare in Vizslas: 1.6% are carriers (n=318). The penetrance signal in the Donner dataset is null; no at-risk dogs in their cohort showed phenotype-confirmed disease. This does not mean the variant is benign, it means the breed’s current carrier pool has not expressed it visibly, but it signals low immediate clinical concern.

Testing exists and is straightforward. Carriers are managed with exercise modulation and heat awareness.

Factor VII Deficiency

Factor VII deficiency in Vizslas is an autosomal-recessive clotting disorder. Affected dogs have reduced Factor VII activity and prolonged clotting times, risking excessive bleeding from trauma or surgery. 0.94% of Vizslas are carriers (n=318). The Donner penetrance data show 6 of 33 at-risk dogs with phenotype confirmation, a maximum penetrance of 18%. Most dogs with two copies do not bleed visibly.

Testing is available and inexpensive. Affected dogs are identified through routine coagulation screening before elective surgery.

What should I feed a Vizsla?

Energy balance is the breed’s defining nutritional challenge. Vizslas were bred for all-day hunting in warm climates and retain that metabolic expectation. A pet Vizsla in a suburban or urban home eating a standard maintenance kibble can become overweight if calories are not matched to actual activity, which compounds latent cardiac risk. Unlike Beagles, which were bred for steady eight-hour hunts and are food-motivated, Vizslas are lean hunters, their body condition should reflect that. Adult maintenance formulations should be measured carefully against body-condition score, not fed ad libitum. Work with your veterinarian to establish a target weight and caloric ceiling.

Vizslas’ lean frame and high metabolism mean skipping fasting days. Unlike some giant breeds that benefit from intermittent fasting protocols, Vizslas on high activity schedules do better with consistent daily calories split into two meals. Consistent meal timing supports stable energy levels in high-activity dogs.

Puppy feeding requires measured calcium intake. Medium-breed puppy formulations with calcium in the 0.8% to 1.2% dry-matter range (NRC 2006) support bone growth without acceleration that strains the growth plates. Vizslas grow to full size by 12 to 14 months; growth-phase nutrition is short but consequential.

What we don’t know

The penetrance of Factor VII deficiency in Vizslas remains unclear. Six of 33 dogs with two copies showed clinical signs in the Donner dataset, but clinical phenotyping is not always complete in screening cohorts. The actual frequency of bleeding episodes in carriers is unknown.

Exercise-induced collapse has not been reported in the Vizsla breed-club literature or the scientific record. The variant exists at low carrier frequency and showed zero phenotype-confirmed cases in the Donner cohort. It is not a breed-defining concern, but the silent-carrier status means screening breeding stock is a reasonable precaution.

Frequently asked questions about Vizslas

How long do Vizslas live? Cited life expectancy is 13.5 years (McMillan 2024 / VetCompass). Breed-club estimates vary with health and activity; individual dogs may live shorter or longer than the cited figure.

What is the most common genetic disease in Vizslas? None of the three Mendelian variants in the top list are common. All three are rare in the breed (carriers at 1.6%, 1.6%, and 0.94% respectively). Vizslas have a relatively clean genetic disease profile compared to many breeds.

Are Vizslas good with children? Vizslas were bred as hunting companions and are affectionate with family. They require significant daily exercise and mental stimulation and are most suitable for active households. They do well with older children; their high energy can overwhelm very young children.

Are Vizslas prone to hip dysplasia? Hip dysplasia data specific to Vizslas are limited in the published record. No Mendelian hip-dysplasia variants appear in the breed’s top list, suggesting low frequency of genetic predisposition. Standard OFA or PennHIP screening at two years of age is reasonable for breeding candidates.

What exercise do Vizslas need? Vizslas are hunting dogs and thrive on 1.5 to 2 hours of vigorous daily exercise. They bond intensely with their owners and are prone to separation anxiety if under-stimulated. They are not suitable for sedentary households.

Do Vizslas have any breed-specific coat or skin issues? No coat or skin conditions appear in the Mendelian variants table for Vizslas. Their short, dense coat requires minimal grooming. Skin health is best maintained through appropriate nutrition, exercise, and standard parasite prevention.

Temperament

What Vizslas tend toward

Tendencies from owner surveys of purebred Vizslas — a leaning across the breed, not a prediction for any one dog. A bar’s strength shows how much of that behavior breed actually explains: for most it’s faint, because the rest is your dog, their training, and the life you give them.

Biddabilitybreed ~18%
biddableindependent
Human Sociabilitybreed ~11%
less sociablehighly sociable
Agonistic Thresholdbreed ~9%
assertivediffident
Environmental Engagementbreed ~9%
high engagementlow engagement
Toy-directed Motor Patternsbreed ~18%
toy-directednot toy-directed
Proximity Seekingbreed ~13%
affectionatealoof
Dog Sociabilitybreed ~8%
less sociablehighly sociable
Arousal Levelbreed ~8%
arousedcomposed
n = 32 dogs · Morrill et al. 2022, Science, Darwin's Ark (CC0)
Owner-reported purebreds; each factor n ≥ 25. "Breed ~%" is the share of this behavior explained by breed.
Names & origins

Other names

The Vizsla is also recorded as Hungarian Pointer, Hungarian Short-Haired Pointer (Vizsla), Hungarian Vizla, Hungarian Vizsla, Hungarian Vizsla unspecifed, Magyar Vizsla, Maygar Vizsla, and Rövidszörü Magyar Vizsla.

Identified as Vizsla (VBO:0201389) in the Vertebrate Breed Ontology (Mullen et al. 2025, CC-BY 4.0) · registry IDs FCI 57 · iDog 421 · VeNom 22528.

For researchers Population genetics, disease models and every recorded condition Open the research detail for the Vizsla
What the atlas says about Vizsla

In the atlas, the Vizsla clusters consistently as Vizsla (100% of the 100 dogs here).

Median lifespan is 13.5 years, about 1.7 years longer than a typical dog of 23.25 kg, an unusually positive longevity for this size.

Genetic dimensions · CanVAS atlas

What the atlas genotypes say about Vizsla

Computed from the 14,478 CanVAS dogs in the Atlas, on an imputed genotype backbone: 77,215 directly typed sites per dog, imputed to 9.67 million variants.

The confidence below counts dogs, not reads: it is the size of the sample, and it is separate from how the genotypes were measured.

Dogs in the Atlas
100Founders
89 from Hayward2016, 7 from Spatola, 4 from Shannon
Genetic diversity
0.31Moderate
Mean heterozygosity across the breed. 51st most diverse of 107 ranked populations.
What does genetic diversity mean?

How varied a breed's gene pool is — the share of gene spots where a typical dog of the breed carries two different versions rather than two identical ones.

How to read it: Higher = more diverse. Among well-sampled breeds it ranges roughly 0.22 (least diverse) to 0.33 (most diverse).

Diversity is a strength, not a verdict on any individual dog. Lower diversity means it's worth paying attention to recessive-risk testing — not that a dog is doomed.

Cluster structure
Splits into two genetic sub-populations
Intra-breed RMS distance: 20.03 · likely working/show-line, regional, or kennel lineage split.
What does within-breed variation mean?

How much individual dogs within the breed differ from each other genetically.

How to read it: Higher = more internal variety among individuals of the breed.

Sensitive to how many dogs of the breed we've sampled.

Related breeds
In the Sporting group
Explore the full lineage map →
VBO foundation stock (breeding records) · AKC breed group
Relatedness is documented lineage + kennel family. Genetic-ancestry distance measures diversity, not kinship, so it isn't used here.
How long they live
13.5years (life expectancy)
95% CI 13.2–13.9 · VetCompass, McMillan 2024, n=2,965. source
What does typical lifespan mean?

The median age dogs of the breed tend to reach.

How to read it: Higher = longer-lived. Compare to longevity-for-size to see whether it's just a size effect.

Drawn from population lifespan records; individual dogs vary widely with care, genetics, and luck.

n = 100 dogs · high sample · CanVAS (Brundage 2026) · Sniff Atlas
A gift to human medicine

Vizslas are a natural model for human disease

Because the same genes cause the same conditions across species, the inherited conditions documented in Vizslas help researchers understand, and work toward treating, the human diseases they model. This is the dog advancing human medicine. The breed models the human disease; it does not have it, and this is not a prediction for your dog.

Human equivalents via OMIA → Mondo / OMIM. Model-of, not identity. The named gene is the canine locus, not a claim that a pet has the human disease.
Documented in OMIA

Every condition recorded in the Vizsla

Beyond the testable carriers above, OMIA's literature catalogue records 7 genetic conditions in the Vizsla, 5 of which have a known human equivalent. This is the documented landscape across all Vizslas ever studied, not a prediction for any one dog.

Online Mendelian Inheritance in Animals (OMIA); Nicholas, Tammen & Sydney Informatics Hub, DOI 10.25910/2AMR-PV70
Documented in the breed's literature is not carrier status and not a forecast for an individual dog. Human equivalents are mapped via Mondo/OMIM. Carrier frequencies (above) are the separately-measured testable subset (Donner 2023).
The data behind this page

Where every number on this page came from.

This page draws on three primary data sources. Carrier frequencies for the Mendelian section come from Donner et al. 2023 (CC-BY-4.0). We grade these data at evidence Limited because the cohort is a direct-to-consumer ascertainment, which biases toward owners who chose to test their dogs. The panel also uses tag-SNP proxies for some variants rather than direct causal-variant assays. Limited is a study-design grade, not a quality grade: the Donner cohort is the largest open canine-genotype dataset in existence and we are grateful for it. We rate the confounding MEDIUM.

Population-genetic dimensions (heterozygosity, intra-breed PCA distance, nearest neighbors, trait-locus frequencies) come from CanVAS (Brundage 2026), harmonized through the Sniff Atlas. The exact release date and verification commit are pinned at the bottom of the page so a researcher can trace a number back to a specific snapshot. The disease-gene-variant graph comes from OMIA (Online Mendelian Inheritance in Animals; Nicholas, Tammen, and the Sydney Informatics Hub at the Sydney School of Veterinary Science, The University of Sydney; retrieved April 2026, DOI 10.25910/2AMR-PV70).

What this page does not yet have. Inheritance modes and per-disease penetrance evidence from Donner 2023 are in the structured data for every variant the panel covers. Mondo, OMIM, Ensembl, and HGNC cross-references live on the gene and disease pages this page links to, rather than being repeated here. What is still absent from this breed page is anything not in the cited panel, and each such gap carries its cause where it appears.

How to cite this page. The computed dimensions on this page are derived from the open Sniff Atlas v1.0.1 (Gehring 2026, doi:10.5281/zenodo.20566358, CC-BY 4.0). Full citation formats including BibTeX, RIS, and CITATION.cff at sniff.world/cite.

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References
  1. Donner J, Freyer J, Davison S, Anderson H, Blades M, Honkanen L, et al. (2023). Genetic prevalence and clinical relevance of canine Mendelian disease variants in over one million dogs. PLOS Genetics 19(2):e1010651. doi:10.1371/journal.pgen.1010651
  2. Brundage DM (2026). CanVAS: A Harmonized and Imputed Canine Variant Atlas. bioRxiv. doi:10.64898/2026.04.13.718238
  3. Nicholas, F.W., Tammen, I., & Sydney Informatics Hub. (2026). Online Mendelian Inheritance in Animals (OMIA) [dataset]. The University of Sydney. https://omia.org. doi:10.25910/2AMR-PV70 (retrieved April 2026).
Last updated
Sources: CanVAS (Brundage 2026) · Donner 2023 · OMIA